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         Basal Ganglia Diseases:     more books (74)
  1. Instrumental Methods and Scoring in Extrapyramidal Disorders
  2. Behavioral Neurology of Movement Disorders (Advances in Neurology)
  3. Extrapyramidal Disorders by Pierre Vinken, George Bruyn, 1987-02-01
  4. Extrapyramidal Disorders in Childhood (International Congress)
  5. Psychotropic Drugs and Dysfunctions of the Basal Ganglia: A Multidisciplinary Workshop by George E. Crane, 1969
  6. Papers on hemiballismus and basal ganglia, 1927-1960 by James Purdon Martin, 1960
  7. Function and Dysfunction in the Basal Ganglia (Studies in Neuroscience) by A. J. Franks, James W. Ironside, et all 1990-05
  8. Basal Ganglia. Association for Research in Nervous and Mental Disease Research Publications Volume 55 by Melvin D., ed Yahr, 1976-01-01
  9. Extrapyramidal Disorders (Journal of Neural Transmission Supplementum)
  10. Hypokinetic Movement Disorders.: An article from: Journal of Neuroscience Nursing by Ellie Borrell, 2000-10-01
  11. Backward inhibition in Parkinson's disease [An article from: Neuropsychologia] by C.L. Fales, Z.F. Vanek, et all
  12. Dopaminergic basis for deficits in working memory but not attentional set-shifting in Parkinson's disease [An article from: Neuropsychologia] by S.J.G. Lewis, A. Slabosz, et all 2005-01

41. Home/news
because the symptoms of Gulf War syndrome strongly resemble early symptoms ofwellstudied degenerative diseases of the basal ganglia like Huntington's

42. Listings Of The World Health Conditions And Diseases
Listings World Health Conditions and diseases Neurological DisordersBrain diseases basal ganglia. Listings World,

43. UMMS Mind Brain And Behavior 1: Connections Of The Basal Ganglia And Associated
are likely to play a role in emotion and cognition (recall the trio of problems thatare so often associated with diseases of the basal ganglia motion, emotion
Glossary Self-Tests Patients Figure List PD: Neurologic Exam StrokeSTOP CNS Overview ... CNS Development Search For:
MBB1 Home
1. Reading 2. Pyramidal and Extrapyramidal Divisions 3. Levels of Motor Integration ... 7. The Ventral Striatum Motor System/Basal Ganglia 5. Connections of the Basal Ganglia and Associated "Extrapyramidal" Structures
We will begin by considering important connections of the basal ganglia that function in movement. Later we will consider related connections that are likely to play a role in emotion and cognition (recall the trio of problems that are so often associated with diseases of the basal ganglia: motion, emotion, and cognition). First, let's see how six major structures - cerebral cortex primarily of frontal lobe, striatum, globus pallidus externus, globus pallidus internus, motor thalamus, and substantia nigra - are interconnected to provide the input to the pyramidal system. [A piece of terminology: striatum = putamen + caudate + ventral striatum; this last structure will be discussed later on]. KEY IDEA: Connections of the basal ganglia are all unilateral.

44. Neuronal Cell Death And Restoration In The Basal Ganglia
Title Neuronal Cell Death and Restoration in the basal ganglia Implications forHuntington's and Parkinson's diseases. Authors Hansson, Oskar. Year 2001.

CEREBROVASCULAR diseases. STROKE. A stroke is defined ischemic injury.basal ganglia are less sensitive to ischemia. Pathologic changes
A stroke is defined as a neurologic deficit with sudden onset due to vascular disease. It is clinical (not pathological) definition.
Cerebral ischemia is a decrease in cerebral blood flow with resultant decrease in cerebral oxygenation. The brain is totally dependent upon aerobic metabolism. It receives 15% of the resting cardiac output and accounts for 20% 0f the oxygen consumption. Hypoxia, regardless of cause, produces a constant pattern of brain injury; however, the injury may be localized or generalized - resulting in two patterns of Acute Ischemic Injury:
  • Ischemic (hypoxic) Encephalopathy - generalized (global) hypoxic injury
  • Cerebral Infarction - localized hypoxic injury with necrosis When blood flow is decreased, followed by subsequent reperfusion, one may see reversible or irreversible brain damage. Reversible Ischemia
    • Focal Ischemia
      • Transient Ischemic Attacks (TIA's) Focal neurologic deficits lasting less than 24 hours. No permanent brain damage results.
      • Theoretically, very severe or prolonged focal ischemia can cause infarction:
  • 46. Dementias And Degenerative Diseases
    result only from diseases of the cerebral cortex, but may also result from diseasesof the See numerous infarcts in the basal ganglia or in the cortex.
    Dementias and
    Degenerative Diseases
    D ementia simply means a general mental deterioration due to organic or psychological factors. This module discusses the pathology of some of the Degenerative Diseases causing dementia. Dementias do not necessarily result only from diseases of the cerebral cortex, but may also result from diseases of the subcortical connections. This is probably true for Alzheimer's Disease, Parkinson's Disease, Huntington's Disease, and the dementia associated with AIDS.
    Alzheimer's Disease (Alz)
    Clinical P rogressive dementia with severe memory loss, especially recent memory. The disease usually develops after age 65. Patients have a slowly progressive dementia which develops over a number of years. The slow onset helps distinguish Alz dementia from more acute onset dementias such as Creutzfeldt-Jakob and AIDS dementia. The disease affects women more than men. Over 50% of demented patients have Alz; more than 600,000 Americans have the disease. There is a familial association for at least 10% of cases. Pathology
    • Gross Mild to moderate cortical atrophy of the frontal, parietal and temporal lobes. There is

    47. Rare Diseases List - Office Of Rare Diseases
    is supported by the Office of Rare diseases in partnership Encephalopathy, familialinfantile, with calcification of basal ganglia and chronic cerebrospinal

    48. L.L. Brown Ph.D.
    Institute to establish the organization of human basal ganglia and to develop a diagnosticprocedure for movement disorders, epilepsy and psychiatric diseases.
    Laboratory Home Page

    Department of Neurology
    and Neuroscience
    Kennedy Bldg, - Room 810
    The Basal Ganglia and Movement Disorders
    The goal of the laboratory is to determine the functional neuroanatomic organization of the basal ganglia, and how the caudate-putamen converts information to carry out an "executive decision." A major project is the search for a functional module of the basal ganglia, a correlate of the cortical "column". We are establishing how strips and patches of axon terminals in the striatum are related to dense patches of mu opioid receptors, and how they may combine inputs to accomplish sensory integration. We use somatosensory stimuli as a probe of basal ganglia organization, and 14C deoxyglucose autoradiography as a measure of neural activity in rat brain.
    In addition, we study the pathology and behavior of a transgenic mouse model of Huntington's disease, to determine abnormal functions of opioid receptors.
    Our studies are relevant to movement disorders, to motor learning, and to psychiatric diseases such as obsessive compulsive disorder and schizophrenia.

    49. Glossary - Nervous System Diseases Health Guide
    The basal ganglia are located immediately below the cerebrum. dementia not a diseaseitself, but group of symptoms that characterize diseases and conditions

    Nervous System Diseases
    Nervous System Disorders... Glossary Index
    Related Resources Within UMM Medical Reference Guide Nervous System Diseases
    acetylcholine - a chemical in the brain that acts as a neurotransmitter. action tremor - a tremor that increases when the hand is moving voluntarily. activities of daily living (ADLs) - personal care activities necessary for everyday living, such as eating, bathing, grooming, dressing, and toileting; a term often used by healthcare professionals to assess the need and/or type of care a person may require. advance directives - documents (such as a Living Will) completed and signed by a person who is legally competent to explain his or her wishes for medical care should he or she become unable to make those decisions at a later time. agitation - a non-specific symptom of one or more physical, or psychological processes in which vocal or motor behavior (screaming, shouting, complaining, moaning, cursing, pacing, fidgeting, wandering) pose risk or discomfort, become disruptive or unsafe, or interfere with the delivery of care in a particular environment. agonist - a drug that increases neurotransmitter activity by stimulating the dopamine receptors directly.

    50. Searchalot Directory For Basal Ganglia
    Macomb Senior High A description of basal ganglia followed by a look at someof the diseases such as sydenhams chorea, huntingtons chorea, cerebral palsy
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    51. :: The Basal Ganglia VII (Advances In Behavioral Biology, 52)
    You are here Conditions diseases Parkinson's Disease The BasalGanglia VII (Advances in Behavioral Biology, 52). Search (books).
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    The Basal Ganglia VII (Advances in Behavioral Biology, 52)
    Catalog: Book
    Manufacturer: Plenum Pub Corp
    Authors: N.Z.) (Cor)/ Faull, Richard L. M. International Basal Ganglia Society Symposium 2001 Waitangi, Louise F. B. Nicholson
    Release Date: 26 September, 2002 Availability: Usually ships within 2 to 3 days List Price: Our Price: Used Price: ThirdPartyNewPrice Price: More Details from Amazon international

    52. II: CNS Vascular Diseases
    II. CNS VASCULAR diseases. vessels arise from proximal major arteries and supplythalamus, internal capsule, and basal ganglia; lenticulostriate and
    Back Home Next II. CNS VASCULAR DISEASES The brain requires 15-20% of normal cardiac output and consumes more oxygen than any other single organ of the body, making it highly vulnerable to injury from hypoxia/ischemia (reduced oxygen/arrest of blood flow). Cerebrovascular events (strokes) result from 1) occlusive vascular disorders, 2) systemic (global) hypoxia/ischemia, and 3) hemorrhage. ANATOMY OF CNS CIRCULATION A) Direct arterial supply to the brain Internal carotid system (supratentorial) a) Small paramedian vessels - perforate base of brain near the midline b) Short circumferential vessels - arise from proximal major arteries and supply thalamus, internal capsule, and basal ganglia; lenticulostriate and thalamostriate arteries (vulnerable to hypertensive changes) c) Long circumferential vessels - cortex and white matter i) Anterior cerebral artery - has good left-right collaterals ii) Middle cerebral artery - receives ~80% of ICA flow iii) Posterior cerebral artery - functionally a part of the basilar system in most people Vertebral-basilar system (infratentorial - except for PCA) a) Small paramedian vessels - perforate midline ventral brainstem b) Short circumferential vessels - lateral brainstem (vulnerable to hypertensive changes) c) Long circumferential vessels - cerebellum and dorsolateral brainstem i) Superior cerebellar artery ii) Anterior inferior cerebellar artery

    53. Clinical Study: 81-N-0010, Regional Cerebral Utilization Of Glucose In Patients
    frontal lobe dementia, atypical Parkinsonian or other basal ganglia disorder because history,examination or laboratory tests that suggest diseases (other than
    Protocol Number: 81-N-0010
    Regional Cerebral Utilization of Glucose in Patients with a Diagnosis of Frontal Lobe Dementia, Atypical Parkinsonian Disorder, and Other Basal Ganglia Disorders
    The Cognitive Neuroscience Section of the National Institute of Neurological Disorders and Stroke proposes to continue its cross-sectional and longitudinal studies of cerebral metabolism in frontal lobe dementias and atypical basal ganglia disorders. These studies include repeated assessments of neuropsychological and brain anatomical and metabolic function in subjects with these important and possibly related brain disorders.
    Sponsoring Institute:
    National Institute of Neurological Disorders and Stroke (NINDS)
    Recruitment Detail
    Type: Active Accrual Of New Subjects Gender:
    Referral Letter Required: No
    Population Exclusion(s): None
    Eligibility Criteria: INCLUSION CRITERIA: Patients with frontotemporal dementia and atypical basal ganglia disorders. Only patients without evidence or history of significant chronic disease who meet standard clinical criteria for the diagnosis of dementia of the Frontotemporal disease type (possible or probable) or atypical basal ganglia disease (e.g., CBD) will be accepted into this protocol. Normal volunteers will be recruited through the NIH normal volunteer office and will undergo the same screening process as the patient group.

    54. Lesch-nyhan2000.msw
    MINOR MESH HEADINGS basalganglia-diseases-pathology; basal-ganglia-diseases-psychology;Lesch-Nyhan-Syndrome-psychology. MAJOR
    go back to the ShuffleBrain main menu
    return to LNS menu
    - the Year 2000 Literature
    web contact: Record 1 of 14 in MEDLINE EXPRESS (R) 2000/11-2000/12 TITLE: Elevated UTP and CTP content in cultured neurons from HPRT-deficient transgenic mice. AUTHOR(S): Brosh-S; Boer-P; Sperling-O; Zoref-Shani-E ADDRESS OF AUTHOR: Felsenstein Medical Research Center, Rabin Medical Center, Petah-Tikva, Israel. SOURCE (BIBLIOGRAPHIC CITATION): J-Mol-Neurosci. 2000 Feb-Apr; 14(1-2): 87-91 INTERNATIONAL STANDARD SERIAL NUMBER: 0895-8696 PUBLICATION YEAR: 2000 LANGUAGE OF ARTICLE: ENGLISH COUNTRY OF PUBLICATION: UNITED-STATES ABSTRACT: Hypoxanthine-guanine phosphoribosyltransferase (EC; HPRT) catalyzes the salvage synthesis of inosine-5'-monophosphate (IMP) and guanosine-5'-monophosphate (GMP) from the purine bases hypoxanthine and guanine, respectively. Complete deficiency of HPRT activity is associated with the Lesch-Nyhan syndrome (LNS), characterized by excessive purine production and severe neurological manifestations. The etiology of the metabolic consequences of HPRT deficiency is clarified, but that of the neurological manifestations is not yet understood. HPRT-deficient mice represent an experimental animal model of LNS. In search for a possible metabolic abnormality in LNS brains, connecting the neurological deficit to HPRT deficiency, the purine and pyrimidine nucleotide content of cultured neurons, prepared from HPRT-deficient transgenic mice, was now determined. The HPRT-deficient neuronal cultures exhibited a significantly elevated content of the pyrimidine nucleotides UTP!

    55. Young_research
    areas of research 1) The functional anatomy of the mammalian basal ganglia; and2) The role of excitatory amino acids in neurodegenerative diseases such as
    Anne B. Young, MD
    Research Profile
    Our laboratory has focused on two primary areas of research: 1) The functional anatomy of the mammalian basal ganglia; and 2) The role of excitatory amino acids in neurodegenerative diseases such as Alzheimer's disease, Huntington's disease and Parkinson's disease. 1) The various nuclei in the basal ganglia have numerous anatomic interconnections that are a challenge to understand on a functional level. Through the combined study of the pharmacology/biochemistry and anatomy of the various pathways, a great deal has been learned over the last twenty years about how pathology in select basal ganglia circuits can lead to disorders as varied as Parkinson's disease, dystonia and Huntington's disease. We are determining the functional relationships between the various basal ganglia nuclei through a combination of lesioning, immunocytochemical, histological, receptor site and transport site autoradiographic techniques and by single and double label in situ hybridization methods of examining gene localization. Using these techniques, we have defined the cell specific expression of glutamate and GABA receptors. Studies on rodent and human basal ganglia are a focus of the lab. With the recent discovery of the Huntington's disease and dystonia DYT1 genes, we have been studying the expression of the genes in rodent and human tissue. In addition, we have demonstrated receptor specific changes in animals transgenic for exon 1 of the HD gene. We are now setting up single cell antisense RNA techniques to examine cell specific receptor and neurotransmitter changes in various animal models of neurodegenerative disease and human neurodegenerative disorders.

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    57. KLUWER Academic Publishers | Basal Ganglia And Thalamus In Health And Movement D
    The major diseases associated with dysfunction of the basal ganglia are Parkinson's,Huntington's, dystonia (abnormal fixed posture with spasms), tremor, and
    Title Authors Affiliation ISBN ISSN advanced search search tips Books Basal Ganglia and Thalamus in Health and Movement Disorders
    Basal Ganglia and Thalamus in Health and Movement Disorders
    Add to cart

    edited by
    Kristy Kultas-Ilinsky
    Dept. of Anatomy and Cell Biology, University of Iowa, Iowa City, USA
    Igor A. Ilinsky
    Dept. of Anatomy and Cell Biology, University of Iowa, Iowa City, USA
    This book presents the latest research by leading researchers on different aspects of the organization and function of the basal ganglia and its output nuclei projections to the thalamus. The major diseases associated with dysfunction of the basal ganglia are Parkinson's, Huntington's, dystonia (abnormal fixed posture with spasms), tremor, and drug-induced dyskinesias. Chapters address both basic sciences, such as the physiology and molecular signaling aspects, and clinical applications, such as lesions and stimulation of the thalamic area of the brain. Contents and Contributors
    Kluwer Academic/Plenum Publishers
    Hardbound, ISBN 0-306-46543-4 March 2001, 388 pp.

    Volume 39 The basal ganglia III Giorgio Bernardi, Malcolm B. Carpenter, G Clinical,and Therapeutic Aspects of Alzheimer's and Parkinson's diseases, Volume 2
    Title Authors Affiliation ISBN ISSN advanced search search tips Bookseries ADVANCES IN BEHAVIORAL BIOLOGY
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    Volumes published in

    59. Sobell Department Studentship Projects 2003
    Little is known of how the latter is achieved and yet abnormal terminationof movement is a core aspect of diseases of the basal ganglia.
    Dr Clinical Motor Neurophysiology Group PhD in human basal ganglia physiology.

    60. Basal Ganglia --- HealthandAge
    Cell loss within the basal ganglia, which may be associated with cell loss in Of allthe chronic diseases associated with 'getting old', osteoarthritis is one!gm=24
    English site German site March 30, 2003

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    Basal ganglia May 24, 2000 Question What is basal ganglion disease?
    Answer The basal ganglia are subcortical, i.e. very close to the thalamus, and are rich in neurotransmitters. The most striking clinical features of basal ganglion disease are those affecting the motor side of the nervous system and comprise of tremor, rigidity, lack of movement and abnormal posture. Basal ganglia may be affected in any diffuse brain disease but degenerative disease causes Parkinson's disease in particular. In this condition, the cause of the degeneration is not known, but there are other causes which mimic the condition, such as atherosclerosis, cerebral anoxia, recurrent head injury and poisonings by certain toxins. Cell loss within the basal ganglia, which may be associated with cell loss in other parts of the brain, may also give rise to progressive supranuclear palsy, multi-system atrophy, and other rarer conditions. Related Links Treatment of Parkinson's disease Please take a moment to give us your comments. For questions about Health matters you may check our

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