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         Basal Ganglia Diseases:     more books (74)
  1. l-dopa impairs learning, but spares generalization, in Parkinson's disease [An article from: Neuropsychologia] by D. Shohamy, C.E. Myers, et all
  2. The roles of sequencing and verbal working memory in sentence comprehension deficits in Parkinson's disease [An article from: Brain and Language] by J. Hochstadt, H. Nakano, et all 2006-06-01
  3. Flanker compatibility effects in patients with Parkinson's disease: Impact of target onset delay and trial-by-trial stimulus variation [An article from: Brain and Cognition] by X.E. Cagigas, J. Vincent Filoteo, et all 2007-04-01
  4. Advances in Neurology: Basal Ganglia and New Surgical Approaches For Parkinson's Disease, Vol. 74 by C. David Marsden, 1997
  5. Advances in Neurology: Basal Ganglia and New Surgical Approaches For Parkinson's Disease, Vol. 74 by C. David Marsden, 1997-01-01
  6. Developments in research: The works of Professor Hirotaro Narabayashi and his associates by Hirotarō Narabayashi, 1988
  7. Brain Biochemistry and Brain Disorders by Philip G. Strange, 1993-01-07
  8. Age-Related Dopamine-Dependent Disorders: International Symposium on Age-Related Monoamine-Dependent Disorders and Their Modulation by Gene and Gend (Frontiers of Neurology and Neuroscience) by M. Segawa, 1995-05
  9. Backus Strikes Back by Jim Backus, Henny Backus, 1984-05
  10. The neuropsychology of ventral prefrontal cortex: Decision-making and reversal learning [An article from: Brain and Cognition] by L. Clark, R. Cools, et all 2004-06-01
  11. Variability in subcortical aphasia is due to variable sites of cortical hypoperfusion [An article from: Brain and Language] by A.E. Hillis, P.B. Barker, et all 2004-06-01
  12. PARKINSONISM: An entry from Macmillan Reference USA's <i>Encyclopedia of Aging</i> by SULTAN DARVESH, 2002
  13. Corticobasal degeneration: An entry from Thomson Gale's <i>Gale Encyclopedia of Neurological Disorders</i> by Richard Robinson, 2005
  14. Is the unilateral lesion of the left substantia nigra pars compacta sufficient to induce working memory impairment in rats? [An article from: Neurobiology of Learning and Memory] by M.I. Bellissimo, I. Kouzmine, et all 2004-09-01

81. Funded Research - MRC
Bacteroidaceae Infections; Barbiturates; basal ganglia; basal gangliaDiseases; Base Sequence; BCG Vaccine; Behavior; Behavior Therapy;
http://fundedresearch.cos.com/cgi-bin/MRC/keyword/main?letter=B

82. UMMS Mind Brain And Behavior 1: Clinical Information About Disorders Of The Basa
Motor symptoms may change as basal ganglia disease progresses As they evolve, diseasesof the basal ganglia may produce excessive movement at first, followed
http://courses.umassmed.edu/mbb1/motor_BG/clinicalinfo_BG.cfm
Glossary Self-Tests Patients Figure List PD: Neurologic Exam StrokeSTOP CNS Overview ... CNS Development Search For:
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1. Reading 2. Pyramidal and Extrapyramidal Divisions 3. Levels of Motor Integration ... 7. The Ventral Striatum Motor System/Basal Ganglia 6. Clinical Information about Disorders of the Basal Ganglia Click here for a table reviewing the spectrum of "extrapyramidal" movement disorders Disorders of Hypermotility (Excessive Movement) In hypermotility disorders, abnormal movements:
Usually decrease during sleep
Usually intensify during stress, anxiety, and excitement
Range in speed from very slow (dystonia) to very fast (tics)
Can be unilateral or bilateral, and focal (involving only certain muscles) or generalized
Four Major Categories of Hypermotility due to Basal Ganglion Disease, and their Symptoms
1. Ceaseless involuntary purposeless movements that occur during rest and during voluntary acts (this group of problems is also called dyskinesias) Major symptoms include:

83. I Abstract /i Proton Magnetic Resonance Spectroscopy Of Basal
Fatigue is a common symptom of neurological diseases that affect basalganglia function. We used proton magnetic resonance spectroscopy
http://www.immunesupport.com/library/showarticle.cfm/id/4357
: Proton magnetic resonance spectroscopy of basal ganglia in chronic fatigue syndrome
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Arthritis Chronic Fatigue ... Weight Loss Free Catalog Advertisement Abstract : Proton magnetic resonance spectroscopy of basal ganglia in chronic fatigue syndrome
ImmuneSupport.com Neuroreport 2003 Feb 10;14(2):225-8 Chaudhuri A, Condon BR, Gow JW, Brennan D, Hadley DM. Clinical Physics. Fatigue is a common symptom of neurological diseases that affect basal ganglia function. We used proton magnetic resonance spectroscopy ( H MRS) to study the metabolic functions of the basal ganglia in chronic fatigue syndrome (CFS) to test the hypothesis that fatigue in CFS may have a neurogenic component. H MRS of left basal ganglia was carried out in eight non-psychiatric patients with CFS and their results were compared to age- and sex-matched healthy asymptomatic healthy controls. A highly significant increase in the spectra from choline-containing compounds was seen in the CFS patient group ( < 0.001). In the absence of regional structural or inflammatory pathology, increased choline resonance in CFS may be an indicator of higher cell membrane turnover due to gliosis or altered intramembrane signalling.

84. Basal Ganglia
basal ganglia In current usage, the phrase 'basal ganglia' means thecaudate nucleus, putamen and globus pallidus. They are functionally
http://www.sci.uidaho.edu/med532/basal.htm
Med.Sci 532 Structure-Function basal ganglia
  • In current usage, the phrase 'basal ganglia' means: the caudate nucleus putamen and globus pallidus They are functionally important, at a minimum, for controlling voluntary movements and establishing postures . When they are altered - say in disorders like Huntington disease or Wilson disease - the person has unwanted movements, such as involuntary jerking movements of an arm or leg or spasmodic movement of facial muscles. The caudate nucleus and putamen along with the interposed anterior limb of the internal capsule are collectively known as the corpus striatum (i.e. striated body) because of their appearance. Similarly, the shape of the putamen and globus pallidus resembles a lens, and they are collectively called the lenticular nucleus
Back to Coronal View Wilson Disease Huntington Disease Tourette Syndrome ... WWAMI

85. Anatomy Of The Brain - Basal Ganglia
basal ganglia. Function Location The basal ganglia is located deep withinthe cerebral hemispheres in the telencephalon region of the brain.
http://biology.about.com/library/organs/brain/blbasalgan.htm
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Anatomy of the Brain
Basal ganglia Function:
  • Controls Cognition
  • Movement Coordination
  • Voluntary Movement
Location: Images:
For additional information see:

86. Neuroscience - Research Programmes
deficits eg impairments of sensorimotor integration and motor timing, in humanbasal ganglia disorders (Parkinson's and Huntington's diseases) are being
http://www.biomed2.man.ac.uk/ns/nsrespro.html
Neuroscience Research Division
Homepage
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Research Programmes
The aim of the Division is excellence in basic and applied neuroscience research. A key objective is to understand more fully the functioning of the CNS and related structures in physiological and pathological states and so develop improved therapies for the treatment of neuronal diseases. To this end, four research programmes have been established which encompass existing strengths in the Division, encourage collaboration between staff and focus future developments. The research in each programme is multidisciplinary, incorporates techniques at the molecular, cellular and tissue level and involves studies in vivo in humans and animals.
  • Movement and its Disorders Other Related Research
  • 1. Movement and its Disorders This programme is aimed at understanding the basic mechanisms of the mammalian neuromuscular system, and the disorders to which it is prone, in order to devise new methods of prevention and treatment.
    The Basal Ganglia and Movement Disorders
    Crossman
    Brotchie , the lab) Sensori-motor Integration
    Experimental investigations of movement deficits e.g. impairments of sensori-motor integration and motor timing, in human basal ganglia disorders (Parkinson's and Huntington's diseases) are being undertaken in collaboration with clinical neuroscientists.

    87. LEFT BASAL GANGLIA LACUNE
    the patient's age should raise the suspicion of pathologic processes such as demyelinatingdiseases, the vasculitidies BILATERAL basal ganglia LACUNAR INFARCTS.
    http://www.netmedicine.com/xray/ctscan/ct40.htm
    LEFT BASAL GANGLIA LACUNE. View image This form of infarct involves small arterioles. In the acute phases of a lacunar infarct, the CT scan may be normal. As the infarct develops it becomes progressively lower in attenuation indicating its chronicity. Due to the dense concentration of neurons in the basal ganglia, a small lacunar infarct may have a dramatic clinical picture. The role of a CT scan in the initial phases of an infarct is essentially to rule out the presence of hemorrhage so that the patient can be anticoagulated. The presence of a very large hemispheric infarct may also preclude the possibility of anticoagulation. Such infarcts may sometimes be noted on the initial CT scan examinations. VENTRICULO-SULCAL PROMINENCE OF AGE. View second image Otherwise known as atrophy. Progressive loss of neurons within the central nervous system with age results in prominence of the ventricles as well as the cisterns when compared with normal age individuals. This does not correspond to the degree of cognitive function. However, the finding of ventriculo-sulcal prominence which is out of proportion to that of the patient's age should raise the suspicion of pathologic processes such as demyelinating diseases, the vasculitidies or chronic cerebro-vascular disease. The small arrow heads point to the sulcal prominence. The large arrows point to the prominent, frontal, temporal horns, the lateral ventricle as well as the prominent fourth ventricle in this order.

    88. HONselect - Parkinson Disease, Secondary
    Browse other medical images in HONmedia? MeSH Broader term(s) Brain diseases. MeSHBroader term(s) Brain diseases Nervous System diseases.
    http://www.hon.ch/HONselect/Selection/C10.228.140.079.862.800.html
    HONselect: Introduction Search Browse Favourites List of Rare Disease add HONselect Contents on "Parkinson Disease, Secondary": MeSH hierarchy and definition Research Articles Web resources Medical Images Medical News Medical Conferences Clinical Trials Wondering about the holiday blues! MeSH Hierarchy English French German Spanish Portuguese MeSH Broader term(s) Diseases
    Nervous System Diseases
    Central Nervous System Diseases
    Movement Disorders
    Parkinsonian Disorders
    Brain Diseases
    Basal Ganglia Diseases
    Parkinsonian Disorders
    Parkinson Disease, Secondary MeSH definition Conditions which feature clinical manifestations resembling primary Parkinson disease that are caused by a known or suspected condition. Examples include parkinsonism caused by vascular injury, drugs, trauma, toxin exposure, neoplasms, infections and degenerative or hereditary conditions. Clinical features may include bradykinesia, rigidity, parkinsonian gait, and masked facies. In general, tremor is less prominent in secondary parkinsonism than in the primary form. (From Joynt, Clinical Neurology, 1998, Ch38, pp39-42)
    Subheadings : complications / diagnosis / embryology / epidemiology / etiology / metabolism / microbiology / surgery / therapy
    MeSH Narrow term(s) MPTP Poisoning Parkinson Disease, Postencephalitic

    89. Medline Record 91356262
    contribute to the neurodegenerative process. Major Indexes basal GangliaDiseases metabolism; Ferritin metabolism; Iron metabolism;
    http://www.aeiveos.com/diet/excess_iron/brain/91356262.html
    Title: Alterations in the levels of iron, ferritin and other trace metals in Parkinson's disease and other neurodegenerative diseases affecting the basal ganglia. Author(s): Dexter DT; Carayon A; Javoy-Agid F; Agid Y; Wells FR; Daniel SE; Lees AJ; Jenner P; Marsden CD Address: Parkinson's Disease Society Research Centre, University Department of Neurology, London, UK. Source: Brain 1991 Aug;114 ( Pt 4):1953-75 Abstract: Major Indexes:
    • Basal Ganglia Diseases [metabolism]
    • Ferritin [metabolism]
    • Iron [metabolism]
    • Parkinson Disease [metabolism]
    • Trace Elements [metabolism]
    Minor Indexes:
    • Aging [metabolism]
    • Atrophy
    • Brain Diseases [metabolism]
    • Brain [metabolism] [pathology]
    • Huntington's Disease [metabolism]
    • Metals [metabolism]
    • Radioimmunoassay
    • Reference Values
    • Supranuclear Palsy, Progressive [metabolism]
    Reagent Names:
    • (Metals)
    • 0 (Trace Elements)
    • 7439-89-6 (Iron)
    • 9007-73-2 (Ferritin)
    Language: English
    Periodical Type: JOURNAL ARTICLE

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