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         Cerebral Edema:     more books (26)
  1. Dynamics of brain edema: Proceedings of the third International Workshop on Dynamic Aspects of Cerebral Edema, Montreal, Canada, June 25-29, 1976
  2. Treatment of Cerebral Edema
  3. Cerebral Edema - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References by ICON Health Publications, 2004-09-16
  4. Brain Edema XIII (Acta Neurochirurgica Supplementum) (v. 13)
  5. Brain Edema XI : Proceedings of the 11th Symposium, Newcastle-Upon-Tyne, United Kingdom, June 6-10, 1999 (Acta Neurochirurgica Supplement) by Eugene Faist, 2001-06-22
  6. Brain Edema XII: Proceedings of the 12th International Symposium, Hakone, Japan, November 10-13, 2002 (Acta Neurochirurgica Supplementum)
  7. Brain Edema VIII: Proceedings of the Eighth International Symposium Bern, June 17-20, 1990 (Acta Neurochirurgica Supplementum 51) by H. J. Reulen, A. Baethmann, et all 1991-04
  8. Recent Progress in the Study and Therapy of Brain Edema
  9. Brain Edema X: Proceedings of the Tenth International Symposium San Diego, California, October 20 - 23, 1996 (Acta Neurochirurgica Supplementum)
  10. Brain Edema IX: Proceedings of the Ninth International Symposium Tokyo, May 16-19, 1993 (Acta Neurochirurgica Supplementum) by K. Baethmann, K. A. Hossmann, et all 1994-04
  11. Cerebral Edema by Louis Bakay, 1965-01-01
  12. Diabetic ketoacidosis prevention starts early. (Cerebral Edema).: An article from: Pediatric News by Doug Brunk, 2003-03-01
  13. Dynamics of Brain Edema: Proceedings of the 3rd International Workshop on Dynamic Aspects of Cerebral Edema, Montreal, Canada, June 25-29, 1976
  14. Treatment of Cerebral Edema (German Edition)

61. High Altitude Cerebral Edema (HACE)
First Previous Index Text. Slide 20 of 27.
http://www.med.wright.edu/em/altitude/sld020.htm

62. High Altitude Cerebral Edema (HACE)
High Altitude cerebral edema (HACE). Symptoms Headache, nausea,vomiting. Signs - Loss of coordination, loss of balance, lassitude
http://www.med.wright.edu/em/altitude/tsld020.htm
High Altitude Cerebral Edema (HACE)
  • Symptoms - Headache, nausea, vomiting
  • Signs - Loss of coordination, loss of balance, lassitude, confusion, drowsiness, slowed mental function, stupor, coma, death
  • Cause
    • Severe brain swelling, chokes off blood supply to brain, compression of brain tissue
    Previous slide Next slide Back to first slide View graphic version

63. Kids' Diabetes Crisis Treatment Can Be Fatal
A common treatment for children in diabetic crisis apparently contributes to a rare but often fatal complication known as cerebral edema, according to a
http://www.wcanews.com/archives/2001/jan/jan2901d.htm
Kids' diabetes crisis treatment can be fatal A common treatment for children in diabetic crisis apparently contributes to a rare but often fatal complication known as cerebral edema, according to a study by researchers at University of California-Davis School of Medicine and Medical Center. The study, the largest and most statistically rigorous examination yet of risk factors for cerebral edema in children with diabetic ketoacidosis, was published in the Jan. 25, 2001 issue of the New England Journal of Medicine Researchers found that bicarbonate, a common treatment for diabetic ketoacidosis, appears to increase the risk of cerebral edema, or swelling of the brain, which can be fatal. In ketoacidosis, toxic levels of acids called ketones build up in the blood. Bicarbonate is often given in the emergency department to reduce these acid levels. "In the past there has been some suspicion that giving bicarbonate can increase risk of cerebral edema, but ours is the first study to show that it can," said Nathan Kuppermann, senior author of the study, a pediatric emergency physician and associate professor of internal medicine at UC- Davis. Cerebral edema can lead to coma, neurological damage, and death. While the complication occurs in only about one percent of diabetic ketoacidosis cases in children, 40-90% of children who develop cerebral edema die, and many others are left with permanent brain damage. In all, cerebral edema accounts for 50-60% of diabetes-related deaths in children.

64. High Altitude Cerebral Edema
Translate this page Anfang Zurück Weiter Ende Index Homepage Text. Folie 28 von 36.
http://www.genista.de/kai/hypoxia/sld028.htm
Anfang Zurück Weiter Ende ... Text Folie 28 von 36

65. High Altitude Cerebral Edema
High Altitude cerebral edema. Symptoms include those of AMS, plus anykind of neurological disorder ataxia, irrationality, hallucinations.
http://www.genista.de/kai/hypoxia/tsld028.htm
High Altitude Cerebral Edema
  • Symptoms include those of AMS, plus any kind of neurological disorder: ataxia, irrationality, hallucinations
  • Can be accompanied by haemmorrhages or thrombosis
  • HACE is life theatening. Untreated, the person will fall to a coma and die within hours to one or two days.
  • Several theories have been proposed, including vasogenic (leakage through the BBB), cytogenic (cell swelling due to lack of oxygen), angiogenesis (severe tissue hypoxia releases factors causing leakage and capillary growth)
Vorherige Folie Nächste Folie Zurück zur ersten Folie Graphik-Version anzeigen

66. All-Net Fulminant Hepatic Failure Hepatic Encephalopathy
specific therapies FHF evaluation identify the etiology ongoing evaluation liverbiopsy complications hepatic encephalopathy cerebral edema bleeding infection
http://www.med.ub.es/All-Net/english/gipage/liver/fhf-16.htm

67. All-Net Fulminant Hepatic Failure Hepatic Encephalopathy
Hepatic encephalopathy and cerebral edema can complicate acute liver failure. Cerebraledema is the leading cause of death in patients with FHF.
http://www.med.ub.es/All-Net/english/gipage/liver/fhf-15.htm

68. Dka
Risk factors for development of cerebral edema in children with diabetic ketoacidosis. Oneof the lifethreatening complications of DKA is cerebral edema.
http://www.uic.edu/pharmacy/services/di/dka.htm
Drug Information Center Risk factors for development of cerebral edema in children with diabetic ketoacidosis Diabetes mellitus is a heterogeneous group of syndromes characterized by an increase in blood glucose. It is caused by an absolute (type 1) or relative (type 2) lack of the hormone, insulin. In type 1 diabetes, which is more common in children, there is a destruction of the b cells of the pancreas by autoimmune antibodies. Approximately 25 to 40 percent of children with type 1 diabetes mellitus will initially be diagnosed upon presentation with ketoacidosis. One of the life-threatening complications of DKA is cerebral edema. Cerebral edema occurs in 1% of children with DKA and is associated with a mortality rate of 40 to 90%. Manifestations of cerebral edema are usually seen several hours after the initiation of therapy. These clinical signs consist of elevated intracranial pressure, alteration and deterioration in alertness or conscious state, diminished reflexes, unequal pupils, or fixed dilated pupils. There is now increasing evidence that subclinical cerebral edema exists in numerous patients treated with insulin and fluids, however it is clinically manifested in few patients. There are few theories hypothesized as to the development of cerebral edema in children with DKA. One of the theories is accumulation of osmolytes in brain cells exposed to hyperosmolar conditions, which can lead to cerebral edema when there is a rapid decrease in extracellular osmolality during treatment. However, the data in this study support another theory, linked to brain ischemia. Factors that can lead to brain ischemia are hypocapnea and dehydration caused by vasoconstriction. In addition, bicarbonate therapy may cause hypoxia via depression of the central nervous system.

69. Diseases Of Brain
Edema, M, Brain, cerebral edema. Edema, M, Brain, cerebral edema. Edema, G, Brain,cerebral edema. Edema, G, Brain, cerebral edema. Encephalocele, G, Brain, Encephalocele.
http://radiology.uchc.edu/eAtlas/nav/msBrain.htm
Diseases of Brain Diagnosis G/M Organ Caption Immature G Brain Immature infant brain Immature G Brain Immature Fetal Brain Agenesis of Corpus Callosum G Brain Agenesis of Corpus Callosum Anencephaly G Brain Anencephaly Anencephaly G Brain Anencephaly Anencephaly G Brain Anencephaly with iniencephaly Anencephaly G Brain Anencephaly Anencephaly G Brain Anencephaly Anencephaly G Brain Anencephaly Arnold Chiari Malformation G Brain Arnold-Chiari Malformation Arnold Chiari Malformation G Brain Arnold-Chiari Malformation Arnold Chiari Malformation G Brain Arnold Chiari Malformation Contusion G Brain Old contusion Encephalocele G Brain Encephalocele Encephalocele G Brain Encephalocele Holoprosencephaly G Brain Semilobar holoprosencephaly Holoprosencephaly G Brain Cyclops / Holoprosencephaly Hydranencephaly G Brain Hydranencephaly Hydrocephalus G Brain Hydrocephalus Hydrocephalus G Brain Hydrocephalus Hydrocephalus G Brain Hydrocephalus Lissencephaly G Brain Lissencephaly Microcephaly G Brain Microcephaly Polymicrogyria G Brain Polymicrogyria Porencephaly G Brain Porencephalic Cyst Tuberous sclerosis G Brain Tuberous Sclerosis Abscess G Brain Abscess Abscess G Brain Cerebellar Abscess Abscess M Brain Abscess Abscess M Brain Abscess Abscess M Brain Abscess Abscess M Brain Tissue surrounding Abscess Meningitis G Brain Meningitis and IVH Meningitis M Brain Meningitis Meningitis M Brain Meningitis

70. Edema Of The Brain
cerebral edema Click on Image to Enlarge it. cerebral edema. •Thecerebral hemispheres are seen from the top with the frontal lobes
http://radiology.uchc.edu/eAtlas/CNS/858.htm

71. Member Sign In
from Neurosurgical Focus. The Neurological Period Benign IntracranialHypertension, cerebral edema, and Steroids. This is, perhaps
http://www.medscape.com/viewarticle/405734_8
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72. Member Sign In
Number 4). Risk Factors for cerebral edema in Children With DiabeticKetoacidosis. Glaser N, Barnett P, McCaslin I, et al. for the
http://www.medscape.com/viewarticle/403997
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73. Diabetes 123 - Treatment Of DKA
diabetes. Furthermore, treating DKA incorrectly can result in cerebraledema and death. cerebral edema A Clinical Emergency! Suspect
http://www.diabetes123.com/d_0n_031.htm
Advertisement E-mail This Article Printer Friendly Format Treatment of DKA Jump to a new section [ Getting Around ] Home Page What's New User's Guide Index Search Site Map [ People ] Parents Kids Adults Friends IDYA Tidbits Parent humor Art Poll Message Board Chat cwdMail Mailing Lists Events Quilt for Life Rufus [ Clinic ] Ask the Diabetes Team Diabetes Basics Diabetes at School Sample 504 Plans Products Marketplace Insulin Pumps la Bomba de Insulina Nutrition Alternate Site Testing Diabetes Dictionary Presentations Diabetes Camps Warning Signs [ Sources ] News and Information Advocacy ADA JDF On-Line Links Regional Info Services [ Feedback ] About Letters to Editor Contribute Advertising Online Store Buy CWD Shirts Logos and Linking Awards Diabetic ketoacidosis, or DKA, is severe, out-of-control diabetes (high blood sugar) that needs emergency treatment. DKA is caused by a profound lack of circulating insulin. This may happen because of illness, taking too little insulin, or getting too little exercise. The body starts using stored fat for energy, and ketone bodies (acids) build up in the blood. DKA also accounts for most hospitalization and is the most common cause of death, mostly due to cerebral edema, in pediatric diabetes. Furthermore, treating DKA incorrectly can result in cerebral edema and death. The Barbara Davis Center for Childhood Diabetes in Denver, Colorado, has prepared an excellent poster for hospital emergency rooms with the correct treatment procedures for DKA and suspected cerebral edema. The guidelines are as follows:

74. April 30, 1998-Vol29n30: What's The Real Cause Of Mountain Sickness?; Research A
who spend time at extreme heightsoxygen deprivation, or hypoxia, resulting inthe potentially lethal brain swelling known as high-altitude cerebral edema.
http://www.buffalo.edu/reporter/vol29/vol29n30/n1.html
VOLUME 29, NUMBER 30 THURSDAY, APRIL 30, 1998 What's the real cause of mountain sickness?; Research at UB points to leaks in blood-brain barrier,
By LOIS BAKER

News Services Editor
The latest high-tech gear can't protect climbers on Mount Everest from one of the hazards feared most by those who spend time at extreme heights-oxygen deprivation, or hypoxia, resulting in the potentially lethal brain swelling known as high-altitude cerebral edema. Research conducted at UB suggests that high-altitude cerebral edema is caused by a disruption in the blood-brain barrier, the specialized layer of cells in cerebral blood vessels that, under normal conditions, allow only certain substances to pass into the brain's circulation. John A. Krasney, professor of physiology and a specialist in hypoxia, presented the findings April 28 at the 66th annual meeting of the American Association of Neurological Surgeons in Philadelphia. Krasney and colleagues in UB's Hermann Rahn Laboratory of Environmental Physiology have developed an animal model using sheep for studying hypoxia and cerebral edema in an effort to learn more about how and why these conditions develop and their effect on cognitive function. "Brain swelling, and also pulmonary edema [fluid in the lungs], is one of the major problems at high altitude and we don't really know the cause of either," Krasney said. "It is not lack of oxygen per se. At high altitude, the brain has an adequate supply of oxygen because blood flow increases to compensate for the lower concentration of oxygen in the air. Yet people still get sick."

75. Msudemail
J Pediatr 1991 Jul;119(1 ( Pt 1))425 cerebral edema causing death inchildren with maple syrup urine disease. Riviello JJ Jr, Rezvani
http://www.mobiletel.com/~abdk/msudemail.html
J Pediatr 1991 Jul;119(1 ( Pt 1)):42-5
Cerebral edema causing death in children with maple syrup urine disease.
Riviello JJ Jr, Rezvani I, DiGeorge AM, Foley CM
Section of Child Neurology, St. Christopher's Hospital for Children, Philadelphia, Pennsylvania.
PMID: 2066857, UI: 91294971
No To Hattatsu 1991 Jan;23(1):71-4
A mild variant case of maple syrup urine disease.
[Article in Japanese]
Ishikawa A, Okayasu T, Miyasaka K, Fukushima N, Takase A, Wagatsuma Y
Department of Pediatrics, Sapporo City General Hospital.
We reported a mild variant case of maple syrup urine disease. He was unable to walk alone at 2 years of age and developed seizures and intermittent ataxia at 5 years of age. Activity of 1-14C-leucine decarboxylase in fibroblasts revealed 40% of normal activity in the boy and 90% in the mother. MRI showed hypo-myelination of white matter and mild atrophy of brain stem and cerebellum. Dietary treatment was not effective for ataxia and brain atrophy. PMID: 1994996, UI: 91136870 Neuroradiology 1998 Jun;40(6):347-54

76. FACTORES DE RIESGO DE EDEMA CEREBRAL EN NIÑOS CON CETOACIDOSIS DIABÉTICA
Translate this page 1. Dunger DB, Edge JA. Predicting cerebral edema during diabetic ketoacidosis. 4.Bullock DG. Management of diabetic ketoacidosis and cerebral edema.
http://www.intermedicina.com/Avances/Pediatria/APE26.htm
FACTORES DE RIESGO DE EDEMA CEREBRAL EN NIÑOS CON CETOACIDOSIS DIABÉTICA Palabras claves: factores de riesgo, edema cerebral, acidosis diabética. La cetoacidosis diabética ocurre en el 25-40% de los niños con diabetes mellitus tipo I recientemente diagnosticada, y puede recurrir luego, por asociarse con otras enfermedades o falta de cumplimiento del tratamiento. El edema cerebral ocurre en el 1% de los casos de cetoacidosis diabética en niños, pero cuando se presenta se asocia con un elevado porcentaje de mortalidad (40-90%). El mecanismo por el cual se produce el edema cerebral es todavía controvertido, por lo que Glaser y col. evaluaron la asociación de esta complicación con características demográficas, características bioquímicas inicial, intervenciones terapéuticas, y cambios en los valores de laboratorio durante el tratamiento. Encontraron una incidencia de edema cerebral de 0,9% de los casos de cetoacidosis diabética. El diagnóstico se basó en deterioro del estado mental acompañado por evidencia radiográfica. La hipocapnia causa vasoconstricción cerebral, deshidratación extrema con su consiguiente hipoxia cerebral, que se exacerba con la terapia con bicarbonato. La hiperglucemia sobreimpuesta altera la barrera hemato-encefálica, que se puede agravar luego de varias horas (4-12 hs) de tratamiento debido a la liberación de sustancias vasoactivas y mediadores de la inflamación. Este mecanismo se puede agravar con tratamientos con altas dosis de insulina y la administración de bicarbonato o grandes volúmenes de solución hipotónica.

77. HallVideo.com :: Treatment Of Cerebral Edema
You are here Video Television Miniseries Treatment of cerebral edema.Search (vhs). Related Items North and SouthNorth and South at $89.98.
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78. American Roentgen Ray Society
patients. Diffuse cerebral edema and hemorrhagic or bland infarctionsin the basal ganglia, thalamus, or cerebellum occur 4 (Figs. 4
http://www.arrs.org/edu/pictorials/0102001/
document.write(""+dayarray[day]+", "+montharray[month]+" "+date+", "+year+"")
American Roentgen
Ray Society
Pictorial Essay
Imaging of Uncommon Cerebral Tropical Infections
Tufail Patankar , Srinivasa Prasad , Girish M. Fatterpekar , Amit Maniar , Mauricio Castillo , Suresh K. Mukherji Department of Radiology, King Edward Memorial Hospital, Parel, Mumbai 400 012, India.
Department of Radiology, University of North Carolina School of Medicine, 3324 Infirmary CB #7510, Chapel Hill, NC 27599-7510. Address correspondence to M. Castillo. AJR (online serial) ;176(6):article 4 Hydatid disease, sparganosis, malaria, dengue fever, and rabies are infections that occur mainly in tropical countries. Although diagnosis is based on clinical and laboratory investigations, imaging studies are performed on patients with hydatid disease and on patients in whom complications are suspected. The purpose of this article is to review the imaging features of these tropical diseases. Hydatid Disease
Echinococcus granulosus and, less frequently, by

79. Selected Altitude Medicine Biliography
High Altitude cerebral edema Other Neurological Problems at Altitude. MailtonAJ, Cymmerman A, Black PM High altitude cerebral edema.
http://www.high-altitude-medicine.com/references.html
Selected Altitude Medicine Bibliography Table of Contents General Altitude Medicine HAPE
General Altitude Medicine Barry PW: Altitude Related Cough, Does It Exist? ISMM News, 7: 3, 8-10 (Summer 1997) Bircher HP, Eichenberger U, Maggiorini M, et al.: Relationship of mountain sickness to physical fitness and exercise intensity during ascent. J Wilderness Med 1994; 5:302-11. Dubowitz G: The Effect of Temazepam on Arterial Oxygen Saturation During Sleep at High Altitude . British Mount Everest Medical Expedition 1994. (This appeared as an abstract on their web page, which no longer exists. I am not sure it was ever published in a journal) Hackett PH, Rennie D: The incidence, importance, and prophylaxis of acute mountain sickness. Lancet 1976; 2:1149-1154. Hackett PH, Hornbein TF: Disorders of High Altitude. Textbook of Respiratory Medicine ; Saunders, Philadelphia 1988; 2:1646-63. Hackett PH, Oelz O: The Lake Louise Consensus on the Definition and Quantification of Altitude Illness. In: Sutton JR, Coates G, Houston CS: Hypoxia and Mountain Medicine; Queen City Printers 1992; 327-330.

80. Study Identifies Risk Factors Part 2
In the past there has been some suspicion that giving bicarbonate can increaserisk of cerebral edema, but ours is the first study to show that it can. .
http://matrix.ucdmc.ucdavis.edu/vol8_no2_mar01/html/edema2.html
continued) "In the past there has been some suspicion that giving bicarbonate can increase risk of cerebral edema, but ours is the first study to show that it can." "The hope," Glaser says, "is that by knowing these risk factors, doctors will be able to monitor the high-risk children better, and pick up the initial signs of this complication before it progresses to a more serious stage." Glaser and Kuppermann report their findings in the Jan. 25 issue of the New England Journal of Medicine. An accompanying editorial praises the study's "rigorous design" and "careful analysis." In the study, the UC Davis researchers reviewed the medical charts of 6,977 children hospitalized for diabetic ketoacidosis at 10 centers, turning up 61 who developed cerebral edema. These children then were compared with two control groups of children with diabetic ketoacidosis who did not develop cerebral edema, using a series of rigorous statistical tests. The analyses revealed that children with diabetic ketoacidosis who have lower initial arterial carbon dioxide readings and higher serum urea nitrogen concentrations are at increased risk for cerebral edema. A smaller increase in serum sodium concentration during therapy also was associated with cerebral edema. Bicarbonate treatment emerged as a fourth risk factor.

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