81. Study Identifies Risk Factors Part 1 Return to Health System Home Page, Matrix. Kristi Oppold, shown with motherTerri, developed diabetic ketoacidosis and cerebral edema five years ago. http://matrix.ucdmc.ucdavis.edu/vol8_no2_mar01/html/edema1.html

Kristi Oppold, shown with mother Terri, developed diabetic ketoacidosis and cerebral edema five years ago. But the Woodland girl, now 13, counts herself lucky: She emerged from her coma with no neurological damage. At first Terri Oppold, a Woodland teacher's aide, thought her eight-year-old daughter, Kristi, had a stomach bug. But when the child vomited nonstop all night long, Oppold suspected something serious. In the emergency room, doctors diagnosed ketoacidosis and right away administered insulin. Yet Kristi grew increasingly agitated and incoherent. By the time a Life Flight helicopter delivered the little girl to UC Davis Medical Center, she had fallen into a coma. Kristi is among the 25 percent to 40 percent of children whose diabetes first presents as ketoacidosis, and among the 1 percent of children with diabetic ketoacidosis who develop cerebral edema. Kristi came out of her coma after 12 hours, with no neurological damage. But many other children aren't as lucky. About one in five youngsters who develop cerebral edema die, and one in four of the children who survive are left with permanent brain damage. The devastating complication ranks as the leading cause of death in children with diabetes mellitus. A new study by UC Davis researchers, however, may help reduce that toll.

82. InteliHealth: Progressive altitude sickness can result in high altitude cerebral edema (HACE),in which the brain fills with fluid, swells and ceases to function properly http://www.intelihealth.com/IH/ihtIH/WSIHW000/9339/28052.html

Entire Site Diseases Conditions Healthy Lifestyle InteliTools Your Health IH Catalog Dental chrome_imgPreload('gifChr_mid_but_home_mo_1','http://img.intelihealth.com/i/C/Chr_mid_but_home-o.gif'); chrome_imgPreload('gifChr_mid_but_shop_mo_2','http://img.intelihealth.com/i/C/Chr_mid_but_shop-o.gif'); chrome_imgPreload('gifChr_mid_but_dental_mo_3','http://img.intelihealth.com/i/C/Chr_mid_but_dental-o.gif'); chrome_imgPreload('gifChr_mid_but_drug_mo_4','http://img.intelihealth.com/i/C/Chr_mid_but_drug-o.gif'); chrome_imgPreload('gifChr_mid_but_askexpert_mo_5','http://img.intelihealth.com/i/C/Chr_mid_but_askexpert-o.gif'); chrome_imgPreload('gifChr_mid_but_medical_mo_6','http://img.intelihealth.com/i/C/Chr_mid_but_medical-o.gif'); chrome_imgPreload('gifChr_mid_but_chats_mo_7','http://img.intelihealth.com/i/C/Chr_mid_but_chats-o.gif'); chrome_imgPreload('gifchr_mid_but_news_mo_8','http://img.intelihealth.com/i/c/chr_mid_but_news-o.gif'); chrome_imgPreload('gifChr_mid_but_privacy_mo_9','http://img.intelihealth.com/i/C/Chr_mid_but_privacy-o.gif'); Advertisement Altitude Sickness What Is It?

83. Diseases Of Brain Aneurysm, G, Brain, Cerebral Aneurysm. Edema, G, Brain, cerebral edema. Edema,G, Brain, cerebral edema. Edema, M, Brain, cerebral edema. Edema, M, Brain, CerebralEdema. http://155.37.5.42/NAV/MSBrain.HTM

Diseases of Brain Diagnosis G/M Organ Caption Immature G Brain Immature infant brain Immature G Brain Immature Fetal Brain Agenesis of Corpus Callosum G Brain Agenesis of Corpus Callosum Anencephaly G Brain Anencephaly Anencephaly G Brain Anencephaly Anencephaly G Brain Anencephaly with iniencephaly Anencephaly G Brain Anencephaly Anencephaly G Brain Anencephaly Anencephaly G Brain Anencephaly Arnold Chiari Malformation G Brain Arnold-Chiari Malformation Arnold Chiari Malformation G Brain Arnold-Chiari Malformation Arnold Chiari Malformation G Brain Arnold Chiari Malformation Contusion G Brain Old contusion Encephalocele G Brain Encephalocele Encephalocele G Brain Encephalocele Holoprosencephaly G Brain Semilobar holoprosencephaly Holoprosencephaly G Brain Cyclops / Holoprosencephaly Hydranencephaly G Brain Hydranencephaly Hydrocephalus G Brain Hydrocephalus Hydrocephalus G Brain Hydrocephalus Hydrocephalus G Brain Hydrocephalus Lissencephaly G Brain Lissencephaly Microcephaly G Brain Microcephaly Polymicrogyria G Brain Polymicrogyria Porencephaly G Brain Porencephalic Cyst Tuberous sclerosis G Brain Tuberous Sclerosis Abscess G Brain Abscess Abscess G Brain Cerebellar Abscess Abscess M Brain Abscess Abscess M Brain Abscess Abscess M Brain Abscess Abscess M Brain Tissue surrounding Abscess Meningitis G Brain Meningitis and IVH Meningitis M Brain Meningitis Meningitis M Brain Meningitis

84. Florida State University College Of Medicine Digital Library cerebral edema Access document; cerebral edema Access document;cerebral edema Access document; cerebral edema Access document. http://fsumed-dl.slis.ua.edu/clinical/emergency/symptoms/edema.htm

Clinical Resources by Topic: Emergency Medicine Edema Clinical Resources Pathology Clinical Guidelines Clinical Trials News ... Miscellaneous Resources See also: General Emergency Medicine Clinical Resources Harrison's Principles of Internal Medicine 15th Ed.-2001: Table of contents Medical Library subscription INFO Chapter 37: Edema Table of contents Introduction: Access document Pathogenesis Access document Clinical Causes of Edema Access document Treatment Access document Differential Diagnosis Access document Localized Edema Access document Edema of Heart Failure Access document Edema of the Nephrotic Syndrome Access document Edema of Acute Glomerulonephritis and Other Forms of Renal Failure Access document Edema of Cirrhosis Access document Edema of Nutritional Origin Access document Other Causes of Edema Access document Distribution of Edema Access document Additional Factors in Diagnosis Access document Approach to the Patient Access document Braunwald-Heart Disease: A Textbook of Cardiovascular Medicine 6th Ed.-2001 (MD Consult): Table of contents Medical Library subscription INFO Chapter 3 - The History: Access document Importance of the History: Access document Cardinal Symptoms of Heart Disease: Access document The History in Specific Forms of Heart Disease: Access document Assessing Cardiovascular Disability Access document Chapter 4 - Physical Examination of the Heart and Circulation: Access document The General Physical Examination: Access document The Cardiac Examination: Access document Dynamic Auscultation: Access document Ferri: Practical Guide to the Care of the Medical Patient 5th Ed.-2001 (MD Consult):

85. Dorlands Medical Dictionary cerebral edema, excessive accumulation of fluid in the brain substance; causes includetrauma, tumor, and increased permeability of capillaries as a result of http://www.mercksource.com/pp/us/cns/cns_hl_dorlands.jspzQzpgzEzzSzppdocszSzuszS

86. COS Expertise Profile infectious emergencies; Pediatric traumatic emergencies; Clinical epidemiology;Pediatric diabetic ketoacidosis and cerebral edema; Multivariable statistical http://myprofile.cos.com/Nathan

University of California, Davis Nathan Kuppermann University of California, Davis School of Medicine Internal Medicine Emergency Medicine Associate Professor Appointed: 2000 Mailing Address Division of Emergency Medicine Department of Internal Medicine/Department of Pediatrics UC Davis Medical Center 2315 Stockton Blvd. Sacramento, California 95817 United States Contact Information Phone: (916) 734-1535 Fax: (916) 734-7950 nkuppermann@ucdavis.edu Qualifications M.P.H., Harvard School of Public Health, Epidemiology and Statistics, 1993 M.D., University of California, San Francisco, Medicine, 1985 B.Sc., Stanford University, Biology, 1981 Expertise and Research Interests Pediatric infectious emergencies; Pediatric traumatic emergencies; Clinical epidemiology; Pediatric diabetic ketoacidosis and cerebral edema; Multivariable statistical modelling. Other Expertise I have experience in coordinating multicenter research in pediatric emergency medicine (PEM). I am currently involved in a PEM collaborative research initiative entitled EMSC Network Development Demonstration Project, sponsored by EMSC and the Maternal and Child Health Bureau. Future Research I am currently involved in clinical epidemilogical studies pertaining to the clinical evaluation of the pediatric trauma patient. These include studies pertaining to the use of computerized tomography for the evaluation of mild-moderate head injury, and abdominal ultrasound evaluation of the pediatric patient sustaining blunt torso trauma.

87. Untitled Chest Pain; 11. cerebral edema; 12. Convulsions/Seizures; 13. cerebral edema (swellingof the brain) is also a common result of severe carbon monoxide poisoning. http://www.emergency.com/co2poisn.htm

FOR IMMEDIATE RELEASE-EMERGENCYNET NEWS SVC.-12/22/94-1030CST THE HIDDEN DANGER OF CARBON MONOXIDE POISONING By C. Staten, EMT-P Chicago, IL. Carbon Monoxide (CO) is a odorless, colorless, tasteless, and insidious gas that takes hundreds of lives each year. The signs and symptoms of carbon monoxide poisoning may include: 1. Headache 2. Dizziness 3. Irritability 4. Confusion/Memory loss 5. Disorientation 6. Nausea and vomiting 7. Abnormal reflexes 8. Difficulty in coordinating 9. Difficulty in breathing 10. Chest Pain 11. Cerebral Edema 12. Convulsions/Seizures 13. Coma 14. Death SIGNS AND SYMPTOMS Commonly, lower-level carbon monoxide patients describe their symptoms as those of a "flu-type" syndrome. They frequently complain of malaise (tiredness), headache, visual disturbances, "runny nose", and may not associate or recognize the symptoms as being the result of CO poisoning. A one month retroactive study of patients reporting to a hospital emergency room with "flu-like" symptoms revealed that almost one quarter of them (23.6%) had a CO level greater than 10% (a diagnosis of carbon monoxide poisoning is generally confirmed by a CO level greater than 10%). Often, several members of the same family or those in a given building will complain of the same symptoms. Children are thought to be more susceptible to carbon monoxide poisoning than adults. Some people may not suspect that CO poisoning is occurring until major symptoms appear. Carbon Monoxide poisoning can mimic gastroenteritis (nausea and vomiting). Other manifestations may cause the appearance of what may appear to be a neurological or psychiatric disorder. High risk groups include infants, the elderly, pregnant women, and anyone with a previous history of cardiac insufficiency or chronic obstructive lung disease.

88. Everest: Physiological Effects High Altitude cerebral edema This occurs in a small portion of peoplewho are afflicted with AMS, usually above 12,000 feet. Cerebral http://www.thetech.org/exhibits/online/everest/about/physiology.htm

Very high altitudes can have serious, and deadly, effects on the human body. Altitude itself is perhaps the greatest threat to the life of any high-altitude climber. As you may know, the higher you climb, the thinner the air becomes. At the top of Mount Everest, the percentage of oxygen in the air is the same as that at sea level (about 20% oxygen). However, atmospheric pressure decreases as altitude increases, and the atmospheric pressure at the summit of Everest is 33% that of sea level. Therefore, there is 66% less oxygen than there is at sea level. Kerosene will not burn. Helicopters cannot fly. Yet somehow, your metabolism is able to continue its chemical conversions inside of you, turning food into heat and energy. At high altitudes breathing becomes a task. Because there is so little oxygen, your body must work harder to pass more air through your lungs. Yet your increased breathing rate taxes your body of more energy. So you must rest. Have you ever felt light-headed after blowing up a balloon? Imagine having that feeling all day long, while trying to climb a hundred flights of stairs, wearing 35 pounds of gear, breathing only through a straw. This may give you an idea of the tremendous challenge that high-altitude climbers face with every single step they take. Altitude sickness occurs most often when there is rapid ascent, and the body does not have sufficient rest time at intermediate elevations to acclimatize. If a person were flown to the top of Everest and released at the summit without oxygen, he or she would die within an hour. Some ways that your body copes with lower oxygen levels are increased production of red blood cells (which carry oxygen throughout the body) and increased production of the enzymes that aid in the release of oxygen to the tissues. In the 1968 Olympics held in Mexico City (elev. 7300 ft/3335m), four of the five endurance track events were won by men who lived at high altitudes.

89. Diabetic Ketoacidosis 6. cerebral edema cerebral edema complicating DKA is an unpredictableand often fatal, but potentially reversible condition. Since http://www.uofodeptpeds.org/diabetic.htm

Department of Pediatrics Affilliated with the University of Ottawa and Children's Hospital of Eastern Ontario Protocols - CHEO Guidelines Diabetic Ketoacidosis Guidelines 1. Initial investigations Consult Diabetes Doctor on call. Blood glucose, pH, pCO2, HCO3, Na, K, Cl, urea, creatinine, urine ketones Calculate Anion gap [Na - (Cl + HC03)] [normal range 12-16] Serum osmolality ([2 x Na ]+ glucose + urea) [normal 275-295 mosm/L] Corrected Na: a 3.5 mmol/L increase in serum glucose depresses serum sodium by 1 mmol/L 2. Monitoring NPO in severe acidosis until nausea subsides - may then have ice chips. Cardiac monitor Cerebral edema is an infrequent but real and dangerous risk for all patients with DKA, and therefore, hourly monitoring of vital signs, level of consciousness, and presence of headache is essential. (Refer to section 6) Accurate intake and output Blood glucose q1h until dextrose added to iv, then q2h (and 1 hour after any changes in insulin dose). May use bedside glucose meter. Blood gas, Na, K, Cl, urea, urine ketones q4h or as clinically indicated. Include lab glucose to confirm the accuracy of the glucose meter.

90. ISI WebServer - Error (404) Paper in peerreviewed journal 1 Losartan versus enalapril on cerebral edema andproteinuria in stroke-prone hypertensive rats. , ELA Blezer, K. Nicolay, HA http://www.isi.uu.nl/Publications/paperlist.php?author=erwin

91. Hypoxia Is The Cause Of Brain Damage In Hyponatremia [Fulltext disorders appear to agree that acute symptomatic hyponatremia, or more precisely,acute water intoxication, imposes the risk of cerebral edema, uncal herniation http://www.ama-assn.org/special/womh/library/readroom/vol_281c/ed90037x.htm

92. Cerebral Edema The summary for this Chinese (Traditional) page contains characters that cannot be correctly displayed in this language/character set. http://www.vghtc.gov.tw:8082/ped/teaching/meta/dka3/sld025.htm

93. Cerebral Edema The summary for this Chinese (Traditional) page contains characters that cannot be correctly displayed in this language/character set. http://www.vghtc.gov.tw:8082/ped/teaching/meta/dka3/sld024.htm

94. 1Up Health > Health Links Directory > Conditions And Diseases: Neurological Diso Conditions and Diseases Neurological Disorders Brain Diseases cerebral edema. ClinicalPoints® Treatments, diagnosis and key points for cerebral edema. http://www.1uphealth.com/links/brain-diseases-cerebral-edema.html

Home Contact Us Privacy Caring For Your Well Being Alternative Medicine Clinical Trials Health News Poisons ... Health Topics A-Z Search 1Up Health Health Directory Addictions Alternative Animal ... Weight Loss By Demography Child Health Teen Health Men's Health Women's Health ... Brain Diseases : Cerebral Edema Description Sites Brain Swelling and Edema An article with a description of these conditions. ClinicalPoints® Treatments, diagnosis and key points for cerebral edema. The Toronto Brain Vascular Malformation Study Group A definition, classifications, pathophysiology and treatment of cerebral edema. WorldMedicus A description of brain edema followed by links to reviews and editorials. Help build the largest human-edited directory on the web. Submit a Site Open Directory Project Become an Editor Parts of the directory made available on 1UpHealth have been modified. External Web site links provided on this site are meant for convenience and for informational purposes only; they do not constitute an endorsement. Search: The information provided herein should not be used during any medical emergency or for the diagnosis or treatment of any medical condition. A licensed physician should be consulted for diagnosis and treatment of any and all medical conditions. Call 911 for all medical emergencies. Links to other sites are provided for information only they do not constitute endorsements of those other sites.

95. Characterization Of CNS White Matter Lesions In Experimental Autoimmune Encephal EL, Nicolay, K., Bar, D., Goldschmeding, R., Jansen, GH, Koomans, HA, and Joles,JA Enalapril prevents imminent and reduces manifest cerebral edema in stroke http://www.imago.uu.nl/imagoch/html/research/nicolay/blezer.htm

Characterization of CNS white matter lesions in experimental autoimmune encephalomyelites (EAE) in the common marmoset using magnetic resonance imaging (MRI) and histology; comparison of data from the animal model with multiple sclerosis Project Title: Characterization of CNS white matter lesions in experimental autoimmune encephalomyelites (EAE) in the common marmoset using magnetic resonance imaging (MRI) and histology; comparison of data from the animal model with multiple sclerosis. Personnel: Dr. ir E.L.A. Blezer Dr. R. van Sluis Prof. dr. ir. K. Nicolay Theme group: Experimental MR spectroscopy and imaging Sponsor: Stichting Vrienden MS Research Collaboration: Biomedical Primate Research Center (Rijswijk, The Netherlands) and MS-MRI Centre (Free University, Amsterdam) Begin: End: Purpose and Scope: The objective of the project is the MRI-guided histopathological analysis of white matter lesions which develop in EAE-affected marmosets and to compare them with lesions in multiple sclerosis (MS) patients. The in-depth comparison of white matter lesions in the patient and the disease model will provide valuable information on the pathological basis of lesion formation in MS and on the relevance of marmoset EAE as a model of the human disease. It will also help to identify characteristics of the different types of MRI detectable lesions to understand the heterogeneity of the lesions. Results Obtained:

96. MD Travel Health - Altitude Sickness - Symptoms, Prevention, And Treatment Severe cases may be complicated by fluid in the lungs (highaltitude pulmonaryedema) or swelling of the brain (high-altitude cerebral edema). http://www.mdtravelhealth.com/illness/altitude_sickness.html

Altitude sickness A recent study indicates that prophylactic inhalation of 125 mcg of salmeterol ( Serevent ) every 12 hours reduces the risk of high-altitude pulmonary edema in those with a prior history of this disorder. See C. Sartori et al, New England Journal of Medicine (2002;346:1631-6) for further information. An alternative for those with a history of high-altitude pulmonary edema is to take oral nifedipine 10 mg every 8 hours or extended-release nifedipine every 12 hours. Other measures to prevent altitude sickness include Ascend gradually or by increments to higher altitudes Avoid overexertion Eat light meals Avoid alcohol If any symptoms of altitude sickness appear, it is essential not to ascend to a higher altitude. If the symptoms become worse or if the person shows signs of cerebral or pulmonary edema, it is essential to descend to a lower altitude . A descent of 500-1000 meters is generally adequate except in cases of cerebral edema, which may require a greater descent. Travelers should not resume their ascent until all symptoms of altitude sickness have cleared. Supplemental oxygen is helpful if available. Acetazolamide, dexamethasone, and nifedipine may all be used to treat altitude sickness as well as prevent it. In most cases, acetazolamide is recommended as prevention, and dexamethasone and nifedipine are reserved for emergency treatment. Nifedipine is preferable to dexamethasone for high-altitude pulmonary edema.

97. Comp_pharm2 Increased cerebral edema surrounding lesion. Several suggested mechanismsof action for reduced cerebral edema including Brain dehydration. http://www.pharmacology2000.com/Central/General_Anesthesia/comp_pharm2.htm

Neurosurgical Considerations : Pharmacological Aspects Example: Supratentorial Intracranial Tumors Supratentorial Intracranial Tumors Meningiomas Gliomas metastatic lesions Compensatory mechanisms to accommodate tumor growth: CSF compartment compression Cerebral vein compression Initially minimal clinical presentations despite elevated ICP, brain structural shifts, and significant mass effect Factors causing significant increases in ICP: Development of a hemorrhagic, necrotic, central volume which may increase in volume rapidly Increased cerebral edema surrounding lesion return to main menu Management of intracranial volume Primary goal: reduction of intracranial volume Pharmacological agents: Corticosteroids; diuretics; anesthetic agents Non-pharmacological interventions: Hyperventilation; BP control; fluid restriction; body position;hypothermia

98. Katalog - Wirtualna Polska Serwis Katalog w Wirtualna Polska S.A. pierwszy portal w Polsce. http://katalog.wp.pl/DMOZ/Health/Conditions_and_Diseases/Neurological_Disorders/

Poczta Czat SMS Pomoc Szukaj.wp.pl: -Katalog -Polskie www -¦wiatowe www -Wirtualna Polska -FTP/Pliki -Grupy dyskusyjne -Encyklopedia -Produkty wp.pl Katalog Katalog ¦wiatowy DMOZ ... Neurological Disorders > Brain Diseases Fakty o Katalogu Pomoc Regulamin Serwis szukaj ... Ostatnio dodane NAWIGACJA Fakty o katalogu Pomoc Regulamin Serwis Szukaj ... FAQ Dodaj stronê Katalog WP Polskie Strony WWW Oferta dla firm WP-HIT ... Wirtualna Polska

A  B  C  D  E  F  G  H  I  J  K  L  M  N  O  P  Q  R  S  T  U  V  W  X  Y  Z

Page 5     81-98 of 98    Back | 1  | 2  | 3  | 4  | 5