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         Hepatocerebral Encephalopathy:     more detail

61. Harding: J Neurol Neurosurg Psychiatry, Volume 58(3).March 1995.pp 320-325.
attention to the possibility of PNDC causing a subacute encephalopathy with prominent NJ,Ellis WG, Washburn ER, Ruebner B. Fatal hepatocerebral syndrome in
http://cloud.prohosting.com/lzambeni/prions/alpersteens.htm
Volume 58(3) March 1995 pp 320-325 Progressive Neuronal Degeneration of Childhood With Liver Disease (Alpers' Disease) Presenting in Young Adults [Papers]
Harding, B N; Alsanjari, N; Smith, S J M; Wiles, C M; Thrush, D; Miller, D H; Scaravilli, F; Harding, A E.
National Hospital for Neurology and Neurosurgery, Queen Square, London, UK. Department of Neuropathology, Department of Clinical Neurophysiology, Department of Clinical Neurology, Derriford Hospital, Plymouth, UK.
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  • [Background] Keywords: progressive neuronal degeneration of childhood; liver disease; Alpers' disease Case reports Graphics Abstract Two unrelated and previously healthy girls, aged 17 and 18, presented with a subacute encephalopathy, visual and sensory symptoms and signs, and prominent seizures that were difficult to control. Brain MRI showed lesions (high signal on T2 weighted images) in the occipital lobes and thalamus; EEG showed slow wave activity with superimposed polyspikes. Inexorable downhill progression led to death in hepatic failure within eight months of onset. Histopathological findings in both patients ((a) chronic hepatitis with prominent bile duct proliferation, fatty change, and fibrosis; (b) in the brain a patchy destruction of the cerebral cortex, predominantly involving striate cortex) were characteristic of progressive neuronal degeneration of childhood with liver diseaseAlpers-Huttenlocher syndromea rare autosomal recessive disorder usually seen in infants and young children.

62. SSIEM Dublin - FINAL PROGRAMME
A newly recognised G protein defect with severe encephalopathy and “curled Mutationsin the deoxyguanosine kinase gene cause hepatocerebral mitochondrial DNA
http://www.ssiem-dublin-2002.com/programme.htm

63. ALCOHOL
5.1 Wernicke's Syndrome (encephalopathy) injury to the midbrain, cerebellum and 5.4Chronic hepatocerebral Disease - scarring of the brain (complication
http://www.stithian.com/Campus_alcohol.htm
ALCOHOL
DRUG TYPE : ALCOHOL CLASSIFICATION : CENTRAL NERVOUS SYSTEM DEPRESSANT ALSO KNOWN AS : BOOZE, A "DOP", JUICE METHOD OF INGESTION : ORAL LEGAL STATUS : MAY BE SOLD TO ANY PERSON OVER THE AGE OF EIGHTEEN YEARS BY A LICENSED LIQUOR OUTLET AVAILABILITY/COST : EASILY AVAILABLE AND CHEAP A Japanese Proverb: "First the man takes a drink, then the drink takes a drink, and then the drink takes the man." BRIEF HISTORICAL FACTS Cambyses, King of Persia in the sixth century B.C., had the dubious distinction of being one of the first alcoholics on record. People of many other early cultures, including the Egyptian, Greek and Roman, made extensive and often excessive use of alcohol. Beer was first made in Egypt around 3000 B.C. The oldest surviving winemaking formulas were recorded by Marcus Cato in Italy almost a century and a half before the birth of Christ. About 800 A.D. the process of distillation was developed by an Arabian alchemist, thus making possible an increase in both the range and the potency of alcoholic beverages. A SOCIAL REALITY Prohibition Prohibition is the forbidding by law of the manufacture, sale, or transportation of alcoholic beverages. Such beverages include beer, gin, rum, whisky and wine. The term Prohibition Era refers to the period of national prohibition that lasted from

64. Neuroradiology
Vascular Dz Children Elderly 1 Hypoxic-Ischemic encephalopathy - varies w pyramidal Cb systems most are inherited Acqiured hepatocerebral Degen - assoc
http://radiology.creighton.edu/Neuroradiology2.html
Intracranial Trauma CRANIOCEREBRAL TRAUMA
MECHANISMS OF INJURY

Projectile Injuries
Nonprojectile Injuries PRIMARY LESIONS CAUSED BY TRAUMA
lesion directly from initial trauma
penetrating injury - av fistula or psuedoaneurysm poss
assoc w superficial temporal or occipital vessels
subgaleal extrusion of brain thru a comminuted skull fx
2. Skull - fx in 60% of acute head injury, 25% of severe none
Fx occurs by Tension rater than compression
plain film evidence of fx is irrelevent in acute care May "grow" in child due to leptomeningeal protrusion Depressed fx - assoc w parenchymal inj, less hem Rhinorrhea or otorrhea - CSF out Temporal Bone Fx - Longitudinal 90%, parallel petrous bone 3. Epidural Hematoma - 1% of head trauma, 10% of fatal injury Lucid Interval - delay before deterioration in 50% usually occur within 24-48 hrs, 20% after neg CT Fx lacerates the mid meningeal art or dural sinus in 95% Biconvex, Strips dural attachments, can't cross suture Poor outcomes II' to delay, may autodecompress thru fx II' herniations common CANNOT cross sutures unless origin is sagital sinus usually at site of inj, subdural can be countercoup

65. Encyclopedia H-Hm (Search FastHealth.com) Encyclopedia H-Hm
amebiasis Hepatic coma Hepatic encephalopathy Hepatic hemangioma D Hepatitis persistentHepatocellular carcinoma hepatocerebral degeneration Hepatolenticular
http://www.jacksonfasthealth.com/encyclopedia/encyclopedia_H-Hm.php
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A-Am
An-Az B-Bm ... Z
H-Hm

H2 Receptor antagonists

Habitual alchohol use

Haemobartonella

Hageman factor
... Dead Links

66. Right Time For Liver Transplants
4. Recurrent hepatic encephalopathy. time is over 100 seconds, serum creatininemore than 3.4mg/dl and coalesce third and forth hepatocerebral degeneration.
http://a531-1.fl.nthu.edu.tw/2001b/liver/time.htm
Right Time for Liver Transplants A. Cholestasic hepatopathy (any one of the conditions being satisfied) 2. Risistant pruritus P rogressing osteopathy 4. Recurrent bacteria cholangitis B. Parenchymatous hepatopathy (any one of the conditions being satisfied) 1. Albumin 2. Prothrombin time more than 3 sec or INR¡Ö1.5 C. Cholestasic hepatopathy and parenchymatous hepatopathy (any one of the conditions being satisfied) Recurrent portal hypertensive bleeding 2. Resistant ascitis Spontaneous bacterial peritonitis Recurrent hepatic encephalopathy Hepatorenal syndrome Severe chronic fatigue Progressing malnutrition Develop retardation Opportune time for liver transplant s in the case of special diseases A. Fulminate liver failure Based on the widely adopted regulations of King's College London, England: Cause d by Acetaminophen ( either one of the conditions being satisfied) Blood power of hydrogen are less than 7.3 (no matter how serious the hepatoencephalopathy degeneration is) Pre- prothrombin time is over 100 seconds serum creatinine more than 3.4mg/dl and coalesce third and forth hepatocerebral degeneration

67. Yougo 1
abscess?…… coma …… encephalopathy…… failure viral……?hepatocerebral syndrome ?
http://plaza.umin.ac.jp/~jaam/98/eiwa.html
o”Å‘O‚̃ŠƒXƒg‚ðŒöŠJ’v‚µ‚Ü‚·BŠF—l‚Ì‚²ˆÓŒ©‚ð‚¨Šñ‚¹‰º‚³‚¢B
i˜A—æF Šw‰ïŽ––±Š ‚Ü‚Å, 1997”N12ŒŽj A B C D ... —pŒêW‚Pi˜a‰pj‚Ö A —pŒêW‚Ri—ªŒêj‚Ö
y Q z QS patternQSƒpƒ^-ƒ“ quadriplegiaŽlŽˆ–ƒáƒ quarantine ŒŸ‰u y X z X-ray sicknessXühŒ y Y z Y-adapter Y-ƒAƒ_ƒvƒ^ Y-piece Y-ƒs[ƒX yZz zero end-expiratory pressure (ZEEP)ŒÄ‹CI––•½ˆ³

68. 1319ch05
Portosystemic encephalopathy (PSE) organic brain disease A severe form ofthe chronic form of PSE is called chronic hepatocerebral disease - brain has
http://www.accd.edu/sac/psychol/kamiller/1319ch05.htm
Introduction to Alcohol and Other Drug Addictions
DAAC 1319/DAAC 1019 Kathryn Miller Any unauthorized use is prohibited by law. Chapter 5: Medical Complications Welcome Getting Started Syllabus Schedule of Lessons ... The Helpdesk Whew! This chapter contains a lot of information about parts of the body, and diseases. I suggest you don't try to read it all at one sitting. Read it slowly and in small chunks. I have included a chapter outline below, but it will not substitute for your careful reading of the chapter. PART I: ADVICE FOR THE MODERATE DRINKER (p. 128)
pregnant women
seizure disorders
diabetes
ALCOHOL - DRUG INTERACTIONS (p. 129) THE BASIS OF ALCOHOL - DRUG INTERACTIONS
1. For moderate drinkers, the MEOS (microsomal ethanol oxidizing system) is
inhibited, so other drugs are not removed as completely or quickly so are still around
in the body.

69. Neuroimaging Aspects Of Chronic Acquired Hepatocerebral Degeneration NOROL BIL D
College and university profiles or web links for over 10 000 colleges and universities.
http://www.pedtoraks.kongresi.net/norolbil/2001/NBD16601.pdf

70. Case 221 --Neuropathology Case
This 77year-old Hispanic-American man with a history of alcoholism and hepatitisC infection suffered severe bouts of recurrent hepatic encephalopathy.
http://path.upmc.edu/cases/case221.html
Case 221 Dementia with Motor Dysfunction in a Patient with Liver Disease
Contributed by David C. Spencer* and Lysia S. Forno+
*Department of Neurology and Neurological Sciences Stanford University and Hospitals
and the +Department of Pathology, VA Palo Alto Health Care System, Palo Alto, CA, USA.
Published on line in February 2000 CLINICAL HISTORY: GROSS AND MICROSCOPIC DESCRIPTION: The only gross abnormalities were slight dilation of the lateral ventricles and a gray discoloration near the junction of cortex and white matter near the motor cortex. On microscopic examination there was striking vacuolation of the deep fronto-parietal cortex and adjacent white matter. ( Fig. 1a , Luxol fast blue-PAS x 2.5). This change was seen to a varying degree in other cortical areas. Prominent pale vesicular astrocyte nuclei (Alzheimer type II glia) were seen in these regions of deep cortex, but were also present in other cortical areas and in the basal ganglia. Some Alzheimer type II glia in the deep cortex contained PAS positive intranuclear inclusions ( Fig. 1b

71. 1Up Health > Diseases & Conditions > H
Hepatic encephalopathy; Hepatic hemangioma; Hepatic ischemia; Hepatic porphyria; Hepatocerebraldegeneration; Hepatolenticular degeneration, see Wilson disease;
http://www.1uphealth.com/health/disease_h.html
1Up Health Home Contact Us Privacy Caring For Your Well Being Alternative Medicine Clinical Trials Health News Poisons ... Health Topics A-Z Search 1Up Health
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72. Searchalot Directory For H
Hemorrhoids (20); Hepatic encephalopathy (3); Hepatitis (88); Hepatocerebralencephalopathy (3); Hepatolenticular Degeneration (14); Hereditary
http://www.searchalot.com/Top/Health/ConditionsandDiseases/H/
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73. Roche Lexikon Medizin (4. Aufl.) - Hepatozerebrale Erkrankung
encephalopathy.
http://www.gesundheit.de/roche/ro15000/r15765.html
hepato zerebrale Erkrankung engl.: hepatocerebral disease
h. Degeneration die W ILSON * Krankheit NOSE (mit Aminoazidurie, Status spongiosus), 2) pseudoulegyrischer Typ (O DA ; S HIRAKI NDO ; S HIRAKI hepatozerebrales Syndrom
h. Syndrom engl.: hepatocerebral syndrome; hyperammonemic encephalopathy hepatozerebrales Syndrom
R
EYE * Syndrom
Verwandte Themen ammoniakalisch Encephalopathia, Enzephalopathie Reye*(-Morgan*-Baral*) Syndrom Wilson* Ableitung

74. ICD-9-CM From Code 570
Pylethrombophlebitis. 572.2 Hepatic coma Hepatic encephalopathy; Hepatocerebralintoxication; Portalsystemic encephalopathy. 572.3 Portal hypertension; 572.4
http://www.cpmc.columbia.edu/homepages/hripcsa/icd9/1tabular570.html
OTHER DISEASES OF DIGESTIVE SYSTEM (570-579)
  • 570 Acute and subacute necrosis of liver
    • Acute hepatic failure
    • Acute or subacute hepatitis, not specified as infective
    • Necrosis of liver (acute) (diffuse) (massive) (subacute)
    • Parenchymatous degeneration of liver
    • Yellow atrophy (liver) (acute) (subacute)
    • Excludes: icterus gravis of newborn (773.0-773.2)
      • serum hepatitis (070.2-070.3)
      • that with:
        • abortion (634-638 with .7, 639.8)
        • ectopic or molar pregnancy (639.8)
        • pregnancy, childbirth, or the puerperium (646.7)
      • viral hepatitis (070.0-070.9)
    • 571 Chronic liver disease and cirrhosis
      • 571.0 Alcoholic fatty liver
      • 571.1 Acute alcoholic hepatitis
        • Acute alcoholic liver disease
      • 571.2 Alcoholic cirrhosis of liver
        • Florid cirrhosis
        • Laennec's cirrhosis (alcoholic)
      • 571.3 Alcoholic liver damage, unspecified
      • 571.4 Chronic hepatitis
        • Excludes: viral hepatitis (acute) (chronic) (070.0-070.9)
        • 571.40 Chronic hepatitis, unspecified
        • 571.41 Chronic persistent hepatitis
        • 571.49 Other
          • Chronic hepatitis:
            • active
            • aggressive
          • Recurrent hepatitis
        • 571.5 Cirrhosis of liver without mention of alcohol

75. Www.naturopatia-e.com -A.N.E.A.oragano Dirigente.scuola
Translate this page in anxious subjects and protracted exposure to endogenous benzodiazepines in hepaticencephalopathy reduce the International symposium hepatocerebral Disorders
http://www.reikiuniversale.it/html/docenti/lorenzo.htm
Ricercatore Accademico Dr. Lorenzo Corsi Esperienze professionali: 1994-1995: Corso di perfezionamento in metodologia di laboratorio su ricerca del farmaco settore farmacologico Dept. Scienze Farmaceutiche Univ. di Modena. In questo periodo il Dott. Corsi ha svolto ricerche riguardanti la caratterizzazione di sostanze ad attivita' benzodiazepino-simile in substrati vegetali ed umani ed il loro ruolo in patologie neurodegenerative. Le ricerche hanno implicato l'uso di tecniche estrattive e di purificazione cromatografiche, prevedendo l'impiego di sistemi HPLC. Inoltre il Dott. Corsi ha acquisito tecniche di Binding radioisotopiche, valutando l'attivita' di tali composti sul sistema Gabaergico, con particolare riferimento al sito di legame per le benzodiazepine e al sito del GABA.
1995-1996: Internato in Farmocologia, Dept. Scienze Farmaceutiche Univ. di Modena.
In questo periodo il Dott. Corsi ha studiato i sistemi recettoriali periferici delle benzodiazepine (Pbr) con riferimento all'espressione del pbr in patologie neoplastiche in particolare nel carcinoma epatocellulare. Sono state perfezionate tecniche di preparazione di membrane epatiche per eseguire binding in frazioni subcellulari (mitocondri, nuclei) ed inoltre sono state approfondite le conoscenze sulle tecniche di binding del Pbr. Le ricerche hanno riguardato anche la valutazione in liquidi biologici di pazienti e tessuti, di putativi ligandi endogeni del Pbr, quali Diazepam Bining Inhibitor (DBI) porfirine e colesterolo.

76. The Everett Files
bilaterally”. Submit that upgoing plantar responses is a typical symptomof hepatic encephalopathy. alkalosis). Compare Liver encephalopathy.
http://ca.geocities.com/xetuyst/2.html
The Everett Files
"Truth Cannot Live on a Diet of Secrets
Withering Within Entangled Lies" H.Michael Sweeney
The Arlene Berry Case
RE: Arlene H. Berry
Date of Death: May 24, 2000
Arlene Berry Photo
This documentation is being restructured. Currently, some of the hyperlinks listed herein may not link to the related sub-information as relevant information is constantly being updated. However, as I continue to develop this page, the links will become active progressively.
INVESTIGATIVE REPORT
Arlene Berry died suddenly and unexpectedly at the age of 41 less than 24 hours after being admitted to the . She presented initially with flu-like symptoms that have since been thoroughly researched and computer traced to the common but unpleasant side effects of a post-operative course of radiation therapy , and chemotherapy , suggestive of iatrogenic hepatic encephalopathy (HE), i.e. liver encephalopathy , or toxic hepatitis . She was transferred to the Sudbury Regional Hospital only hours before she died, where a new head CT was performed. It shows attenuating collections of blood suggestive of multiple Blood Clots , such as seen in

77. Access 10,000 + Universities And Colleges At Universities.com - (campus And Dist
12); Hepatic encephalopathy@ (3); Hepatitis@ (85); Hepatocerebralencephalopathy@ (3); Hepatolenticular Degeneration@ (14); Hereditary
http://www.studentbody.com/Directory/Health/Conditions_and_Diseases/H/

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  • 78. NSW Health - ICD-9-CM 520-579
    Pylethrombophlebitis; 572.2 Hepatic coma Hepatic encephalopathy Hepatocerebralintoxication Portalsystemic encephalopathy; 572.3
    http://www.health.nsw.gov.au/public-health/icd/520-579.htm
    9. DISEASES OF THE DIGESTIVE SYSTEM (520-579)
    DISEASES OF ORAL CAVITY, SALIVARY GLANDS, AND JAWS (520-529)
    520 Disorders of tooth development and eruption
    • Anodontia Absence of teeth (complete) (congenital) (partial) Hypodontia Oligodontia Excludes : acquired absence of teeth (525.1)
    • Supernumerary teeth Distomolar Fourth molar Mesiodens Paramolar Supplemental teeth Excludes : supernumerary roots (520.2)
    • Abnormalities of size and form Concrescence of teeth Fusion of teeth Gemination of teeth Dens evaginatus Dens in dente Dens invaginatus Enamel pearls Macrodontia Microdontia Peg-shaped [conical] teeth Supernumerary roots Taurodontism Tuberculum paramolare Excludes : that due to congenital syphilis (090.5) tuberculum Carabelli, which is regarded as a normal variation
    • Mottled teeth Dental fluorosis Mottling of enamel Nonfluoride enamel opacities
    • Disturbances of tooth formation Aplasia and hypoplasia of cementum Dilaceration of tooth Enamel hypoplasia (neonatal) (postnatal) (prenatal) Horner's teeth Hypocalcification of teeth Regional odontodysplasia Turner's tooth Excludes : Hutchinson's teeth and mulberry molars in congenital syphilis (090.5) mottled teeth (520.3)

    79. Postgraduate Medicine: Primary Care Guide To Myoclonus And Chorea
    Polycythemia vera Neuroacanthocytosis Chorea gravidarum Acquired hepatocerebraldegeneration. or action myoclonus as a sequel to hypoxic encephalopathy.
    http://www.postgradmed.com/issues/2000/10_00/caviness.htm
    Primary care guide to myoclonus and chorea
    Characteristics, causes, and clinical options
    John N. Caviness, MD VOL 108 / NO 5 / OCTOBER 2000 / POSTGRADUATE MEDICINE CME learning objectives
    • To become familiar with the major diagnostic categories of myoclonus
    • To recognize the major clinical characteristics of Huntington's disease
    • To learn the common causes of chorea and to recognize characteristics of various types of choreic disorders
    The author discloses no financial interests in this article. This is the third of four articles on hyperkinetic movement disorders This page is best viewed with a browser that supports tables. Preview : Myoclonic and choreic disorders encompass a wide range of manifestations, from simple jerking during sleep in neurologically normal persons to wild, potentially injurious flailing in patients with ballismus. Recognition of the type and cause of the disorder is the first step toward helping patients achieve better functioning when possible. Dr Caviness reviews classification of myoclonus and chorea and discusses appropriate evaluation and the available treatment options.
    Caviness JN. Primary care guide to myoclonus and chorea: characteristics, causes, and clinical options. Postgrad Med 2000;108(5):163-72

    80. Untitled
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