1. EMG/NEUROMUSCULAR DISORDERS FELLOWSHIP clinical and laboratory research in ALS, demyelinating and hereditary neuropathies, mysasthenia gravis, and myopathies. http://www.phymac.med.wayne.edu/departments/Neurology/EMG.html

Neuromuscular Disorders Fellowship Wayne State University Harper Hospital Directors: James Selwa , M.D. Richard A. Lewis, M.D. Address of director: Department of Neurology 6E-University Health Center 4201 St. Antoine Detroit, MI 48201 Faculty and Interests: James Selwa , M.D. EMG, neuromuscular diseases, diabetic neuropathies, computer applications to clinical neurology Richard A. Lewis, M.D. Inherited and acquired neuropathies, ALS, myasthenia gravis, EMG Michael Shy , M.D. Genetic demyelinating neuropathies in humans and animal models, immune mediated neuropathies, EMG Robert P. Lisak, M.D. Clinical and basic research in neuroimmunologic diseases, myasthenia gravis Sawsan Abu-Shakra, M.D. EMG, mechanisms of muscle cell death, role of immediate early genes in cell death, apoptosis Clare Braun Hashemi, M.D. Neuromuscular and autonomic disorders, EMG M. Maher Fakhouri, M.D. EMG, pain John Kamholz , M.D., Ph.D. Genetic and molecular biologic aspects of disorders of central and peripheral myelin James Garbern , M.D., Ph.D. Neurogenetics; Peripheral neuropathy in Pelizaeus-Merzbacher disease; genetic disorders affecting myelin

2. Neurodegeneration seropositive and seronegative patients with mysasthenia gravis reversibly blocks currents through nicotinic http://www.mh-hannover.de/kliniken/neurologie/forschung/Publikation.html

Stand: 08/2001 Publikationen PD Dr. J. Bufler Bufler J , Pitz R, Czep M, Wick M, Franke C. Purified IgG from seropositive and seronegative patients with mysasthenia gravis reversibly blocks currents through nicotinic acetylchuline receptor channels. Ann.Neurul. 1998;43:458-64. Bufler J , Choi GC, Franke C, Schepp W, Prinz C. Vultage-gated Ca2+ currents in rat gastric enterochromaffin-like cells. Am.J.Physiul 1998;274:C424-C429. Bufler J , Heckmann M, Jahn K, Franke C. Distribution of desensitization time constants of mouse embryonic-like nicotinic and homomeric GLUR6 glutamate receptor channels. Neurosci.Lett. 1997;221:173-76. Bufler J , Kahlert S, Tzartos S, Toyka KV, Maelicke A, Franke C. Activation and blockade of mouse muscle nicotinic channels by antibodies directed against the binding site of the acetylchuline receptor. J.Physiul 1996;492 ( Pt 1):107-14. Bufler J , Franke C, Parnas H, Dudel J. Open channel block by physostigmine and procaine in embryonic-like nicotinic receptors of mouse muscle. Eur.J.Neurosci. 1996;8:677-87. Bufler J , Wilhelm R, Parnas H, Franke C, Dudel J. Open channel and competitive block of the embryonic form of the nicotinic receptor of mouse myotubes by (+)-tubocurarine. J.Physiul 1996;495 ( Pt 1):83-95.

3. Neurobiology (Biology 405) Chemical Messengers; Postsynaptic Receptors; Diseases of Transmissionmysasthenia gravis. Sensory Perception Systems Receptor Physiology; http://www.manhattan.edu/science/biology/courses/biol405.html

4. Neurology Special interests nerve and muscle disease, movement disorders, dystonia, mysasthenia gravis. http://www.delawaretoday.com/top_docs/neurology.cfm

Neurology *Charles Bean 1228 N. Scott St., Wilmington, DE 19806, 656-2527. Medical school: Bowman-Gray School of Medicine, Wake Forest University. Residencies: Children's Hospital of Boston; Massachusetts General Hospital, Boston; National Institute of Health, Bethesda, MD. Board certified in pediatrics and neurology with special competence in child neurology. In practice for 23 years. Special interests: seizure disorder and epilepsy, movement disorders. *Lanny Edelsohn 1228 N. Scott St., Wilmington DE 19806, 656-2527. Medical school: Hahnemann. Residency: Harvard. Board certified in neurology. In practice for 25 years. Special interests: Alzheimer's disease, Parkinson's disease, MS, stroke. *Alan J. Fink 1228 N. Scott St., Wilmington, DE 19806, 656-2527. Medical school: SUNY at Buffalo. Residency (internal medicine): SUNY Buffalo. Residency (neurology): Yale University. Board certified in neurology. In practice 23 years. Special interests: neuro-ophthalmology and neuro-radiology. Stephen F. Penny The Center for Neurology, Neurosurgery and Pain Management, 1093 S. Governors Ave., Dover, DE 19904, 678-8100. Medical school: Jefferson. Residency (neurology): National Naval Medical Center, Bethesda, MD. EMG/ Fellowship (neuromuscular disease): Duke University Medical Center, Durham, N.C. Board certified in neurology with added qualifications in clinical neurophysiology. In practice for 4 years. Special interests: nerve and muscle disease, movement disorders, dystonia, mysasthenia gravis.

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12. 1996 Exam 2 Questions true false. 8) mysasthenia gravis is an autoimmune disease that canbe effectively treated with LDOPA (Dopar, Larodopa). true false. http://www.courses.vcu.edu/ptxed/m2/1996/96-2_q1-12.htm

1996 QUESTIONS Time is running out..... Plan for 1 min per question Indicate whether the following statements are true or false based on the material you have learned.. The primary neurotransmitter involved in neurohumoral transmission in both sympathetic ganglia and parasympathetic ganglia is nicotine. true false All of the following actions are associated with activation of muscarinic receptors: contraction of bronchial smooth muscle, decrease in heart rate and increase in sweat secretion. true false Stimulation of beta1-adrenoceptors in the uterus can delay premature labor. true false Preganglionic fibers of the autonomic nervous system are myelinated, whereas the postganglionic fibers are non-myelinated true false The ganglia of the parasympathetic nervous system are principally distributed diffusely in the walls of the tissues or organs they innervate. true false The enzyme responsible for the conversion of dopamine to norepinephrine is the rate limiting step in the synthesis of norepinephrine. true false Neuronal uptake (Uptake 1) at the adrenergic neuroeffector junction can be inhibited by reserpine (Sandril) to cause chronic depletion of transmitter stores.

13. Untitled She gave up live performing because it was tough on her husband, Roger Smith,who suffers from mysasthenia gravis, a debilitating muscular disease. http://www.kittenwhip.net/reviews/march1.html

Debut: It’s A First For Ann-Margret The Philadelphia Daily News March 1, 2001 Stu Bykofsky While she’s sexy singer/dancer and frothy actress, Ann-Margret over the years has demonstrated some serious acting chops in film and on TV. So why did she choose the relatively lightweight fole as the madam in “The Best Little Whorehouse In Texas” for her stage debut? The answer, she says, is that over the years she has been offered substantial roles on Broadway, but “I just couldn’t see myself tearing myself away from family and friends and our home and our pets. We’ve got six cats and a dog who thinks she’s a cat, a Matese.” In fact, the last time the actress, who will turn 60 next month, appeared anywhere live was New Year’s Eve six years ago, when she starred at Caesars in Atlantic City. She gave up live performing because it was tough on her husband, Roger Smith, who suffers from mysasthenia gravis, a debilitating muscular disease. “It was too hard on Roger,” she says. “We were doing one-nighters at one point. And he’s like – you know the term that President Truman used, the buck stops here? That’s they way it is with my husband. Everyone goest to him, and we’ve got quite a big group.” The stage play is a little bit different. Although it is scheduled, at this writing, for 28 cities, Ann-Margret and the cast will get the summer off, and A-M will travel with her husband, 81-year old mother, Anna, and Missy the Maltese. (The show opens Tuesday for a one-week run at the Merriam.)

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15. Nerve Conduction Studies For The Foot And Ankle from the body. If you are on medication for mysasthenia gravis pleasealso notify the technician before testing. Please wear loose http://www.whymyfoothurts.com/conditions/nerveconduction.html

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