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Amnesia is a memory condition in which memory is disturbed. In simple terms it is the loss of memory. The causes of amnesia are organic or functional. Organic causes include damage to the brain, through trauma or disease, or use of certain (generally sedative) drugs. Functional causes are psychological factors, such as defense mechanisms. Hysterical post-traumatic amnesia is an example of this. Amnesia may also be spontaneous, in the case of transient global amnesia. This global type of amnesia is more common in middle-aged to elderly people, particularly males, and usually lasts less than 24 hours. Another effect of amnesia is the inability to imagine the future. A recent study published online in the Proceedings of the National Academy of Sciences shows that amnesiacs with damaged hippocampus cannot imagine the future. This is because a normal human being, imagining the future, uses past experiences to construct a possible scenario. ... Read more

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Chapters: Fugue State, Transient Epileptic Amnesia, Transient Global Amnesia, Anterograde Amnesia, Psychogenic Amnesia, Repressed Memory, Tip of the Tongue, False Memory Syndrome, Childhood Amnesia, Korsakoff's Syndrome, Post-Traumatic Amnesia, Sywald Skeid, Hyperthymesia, Blackout, Twilight Sleep, Retrograde Amnesia, the Seven Sins of Memory, Absent-Minded Professor, Source Amnesia, Bromism, Absent-Mindedness, Lacunar Amnesia, Memory Distrust Syndrome, Prosopamnesia, Source-Monitoring Error, Selective Memory Loss, Ribot's Law, Memory Disorder. Source: Wikipedia. Pages: 159. Not illustrated. Free updates online. Purchase includes a free trial membership in the publisher's book club where you can select from more than a million books without charge. Excerpt: Transient epileptic amnesia (TEA) is a rare but probably underdiagnosed neurological condition which manifests as relatively brief and generally recurring episodes of amnesia caused by underlying temporal lobe epilepsy. Though descriptions of the condition are based on fewer than 100 cases published in the medical literature, and the largest single study to date included 50 people with TEA, TEA offers considerable theoretical significance as competing theories of human memory attempt to reconcile its implications. A person experiencing a TEA episode has very little short-term memory, so that there is profound difficulty remembering events in the past few minutes (anterograde amnesia), or of events in the hours prior to the onset of the attack, and even memories of important events in recent years may not be accessible during the amnestic event (retrograde amnesia). Some people report short-lived retrograde amnesia so deep that they do not recognize their home or family members, though personal identity is preserved. The amnestic attack has a sudden onset. Three-fourths of cases are reported upon awakening. In attacks that begin when an individual is fully alert, olfacto...More: http://booksllc.net/?id=18716068 ... Read more

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Targeted to patients, their families and allied health students, The “Gale Encyclopedia of Neurological Disorders” provides in-depth coverage of neurological diseases and disorders, including stroke, multiple sclerosis, Parkinson disease, Tourette Syndrome, Alzheimer's disease, cerebral palsy, vertigo, amnesia and epilepsy. Related topics include communication aids, electric personal assistive mobility devices, medications for treating neurological diseases and conditions, understanding the needs of Alzheimer patient caregivers and more. This two-volume set provides an alternative to resources that either fail to explore neurological disease in any depth and or do so at a level not appropriate for students and general readers.

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This digital document is an article from Southern Medical Journal, published by Southern Medical Association on August 1, 2004. The length of the article is 1815 words. The page length shown above is based on a typical 300-word page. The article is delivered in HTML format and is available in your Amazon.com Digital Locker immediately after purchase. You can view it with any web browser.

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Title: Marijuana-induced transient global amnesia.(Case Report)
Author: Prem C. Shukla
Publication: Southern Medical Journal (Refereed)
Date: August 1, 2004
Publisher: Southern Medical Association
Volume: 97Issue: 8Page: 782(3)

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This digital document is a journal article from Neuropsychologia, published by Elsevier in 2006. The article is delivered in HTML format and is available in your Amazon.com Media Library immediately after purchase. You can view it with any web browser.

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In a previous study, we investigated the relationship between the disorders of both episodic memory and working memory in the acute phase of transient global amnesia (TGA). Since executive functions were spared, another dysfunction may be responsible for the binding and maintenance of multimodal informations and contribute to the encoding disorders observed in some patients [Quinette, P., Guillery, B., Desgranges, B., de la Sayette, V., Viader, F., & Eustache, F. (2003). Working memory and executive functions in transient global amnesia. Brain, 126, 1917-1934.]. The aim of this present study was to assess the functions of binding and maintenance of multimodal information during TGA and explore their involvement in episodic memory disorders. We therefore conducted a more thorough investigation of working memory in 16 new patients during the acute phase of TGA using two tasks designed to assess the binding process and both dimensions of the maintenance, namely the active storage and the memory load ability. We also investigated the nature of the episodic memory impairment in distinguishing between the performance of patients with preferential encoding deficits and those of patients with preferential storage disorders on the episodic memory task. This distinction was closely related to the severity of amnesia, i.e. an encoding disorder was observed rather in the early phase of TGA. The results showed that while the functions of binding and maintenance of multimodal information were intact in patients with storage disorders, they were impaired in the case of encoding deficits. These results are interpreted in the recent framework of episodic buffer proposed by Baddeley [Baddeley, A. D. (2000). The episodic buffer: A new component of working memory? Trends in Cognitive Sciences, 4, 417-423] that represents an interface between working memory and episodic memory. ... Read more