21. Re: Bergers Disease kidney disorders message board. Re bergers disease. In Reply tobergers disease posted by Nancy on July 12, 2000 at 174323 http://www.healthboards.com/kidney-disorders/457.html

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22. Re: IgA Nephropathy hello, id like more info on the bergers disease testing group who claim to havea new treatmnent that they were sending to people who wanted to try it. http://www.he.net/~brumley/renal/adminmessages/1289.html

Re: IgA Nephropathy Follow Ups Post Followup Administrators Forum FAQ Posted by john on January 09, 19103 at 16:59:41: In Reply to: Re: IgA Nephropathy posted by john jeansonne on January 09, 19103 at 16:58:07: : hello, id like more info on the bergers disease testing group who claim to have a new treatmnent that they were sending to people who wanted to try it. can anyone tell me what it was? or if its still being sent out? thank you,,, my email is orenjohn@bayou.com Follow Ups: Post a Followup Name: E-Mail: Subject: Comments: : : hello, id like more info on the bergers disease testing group who claim to have a new treatmnent that they were sending to people who wanted to try it. can anyone tell me what it was? or if its still being sent out? thank you,,, : my email is orenjohn@bayou.com Optional Link URL: Link Title: Optional Image URL: Follow Ups Post Followup Administrators Forum FAQ

23. Re: IgA Nephropathy hello, id like more info on the bergers disease testing group who claim to havea new treatmnent that they were sending to people who wanted to try it. http://www.he.net/~brumley/renal/adminmessages/1288.html

Re: IgA Nephropathy Follow Ups Post Followup Administrators Forum FAQ Posted by john jeansonne on January 09, 19103 at 16:58:07: In Reply to: Re: IgA Nephropathy posted by ROBERT M. USTIANOWSKI on October 25, 19102 at 16:24:50: hello, id like more info on the bergers disease testing group who claim to have a new treatmnent that they were sending to people who wanted to try it. can anyone tell me what it was? or if its still being sent out? thank you,,, Follow Ups: Re: IgA Nephropathy john Post a Followup Name: E-Mail: Subject: Comments: : hello, id like more info on the bergers disease testing group who claim to have a new treatmnent that they were sending to people who wanted to try it. can anyone tell me what it was? or if its still being sent out? thank you,,, Optional Link URL: Link Title: Optional Image URL: Follow Ups Post Followup Administrators Forum FAQ

24. Virtual Hospital: Radiology Resident Case Of The Week: Hypothenar Hammer Syndrom Clinical Sx 34year-old male smoker with ischemic fourth and fifthdigits of the hand with possible diagnosis of bergers disease. http://www.vh.org/adult/provider/radiology/RCW/021397/021397.html

For Providers Radiology Resident Case of the Week: February 13, 1997 Hypothenar Hammer Syndrome Ronald W. Fuller M.D. Peer Review Status: Internally Peer Reviewed Clinical Sx: 34-year-old male smoker with ischemic fourth and fifth digits of the hand with possible diagnosis of Bergers disease. Etiology/Pathophysiology: Hypothenar hammer syndrome results from repetitive blunt trauma to the palm of the hand. The position of the ulnar artery in the hypothenar eminence crossing the hamate bone makes it vulnerable to repetitive occupational or recreational trauma that may result in aneurysm formation or ulnar artery thrombosis, with the aneurysm occasionally serving as a source for digital emboli. Imaging: The arterial lesion may appear angiographically as spasm, occulsion, aneurysm, or a combination. Arterial narrowing may result from arterial spasm, extrinsic compression, or subintimal hematoma. Intraluminal filling defects may represent intraluminal thrombosis or intimal flaps. Intimal flaps appear as thin lucent strips in the contrast column. Pseudoaneurysms are a focal widening of the lumen as a result of damage to the arterial media. DDX: Emboli from different etiologies and atheroscerotic disease that can usually be differentiated by history.

25. Www.usrds.org/resguide/appendices/appendix_g/means_frequencies/medevid.txt Syndrome 1 0.0 5834Z ANTIGBN GLOMERULONEPHRITIS 3,058 1.2 5836A Tubular necrosis(no recovery) 2,094 0.8 58381B IgA nephropathy, bergers disease 227 0.1 http://www.usrds.org/resguide/appendices/appendix_g/means_frequencies/medevid.tx

26. Www.usrds.org/resguide/appendices/appendix_g/means_frequencies/patients.txt Tubular necrosis (no recovery) 244 0.0 5837Z NEPHRITIS OR NEPHROPATHY WITH RENALMEDULLARY LESION 1,872 0.2 58381B IgA nephropathy, bergers disease 204 0.0 http://www.usrds.org/resguide/appendices/appendix_g/means_frequencies/patients.t

27. Virtual Hospital: Radiology Resident Case Of The Week: Hypothenar Hammer Syndrom History Clinical Sx 34 year old male smoker with ischemic fourth and fifthdigits of the hand with possible diagnosis of bergers disease. http://www.janela1.com/vh/docs/v0000990.htm

Radiology Resident Case of the Week Hypothenar Hammer Syndrome February 13, 1997 Ronald W. Fuller M.D. Peer Review Status: Not reviewed History Clinical Sx: 34 year old male smoker with ischemic fourth and fifth digits of the hand with possible diagnosis of Bergers disease. Etiology/Pathophysiology Hypothenar hammer syndrome results from repetitive blunt trauma to the palm of the hand. The position of the ulnar artery in the hypothenar eminence crossing the hamate bone makes it vulnerable to repetitive occupational or recreational trauma which may result in aneurysm formation or ulnar artery thrombosis, with the aneurysm occasionally serving as a source for digital emboli. Pathology: Miscellaneous Imaging The arterial lesion may appear angiographically as spasm, occulsion, aneurysm, or a combination. Arterial narrowing may result from arterial spasm, extrinsic compression, or subintimal hematoma. Intraluminal filling defects may represent intraluminal thrombosis or intimal flaps. Intimal flaps appear as thin lucent strips in the contrast column. Pseudoaneurysms are a focal widening of the lumen as a result of damage to the arterial media. DDX Emboli from different etiologies and atheroscerotic disease which can usually be differentiated by history.

28. Changes International Bulletin Board Provided By Independent Executive Suzy Caru help. Evan 133655 5/01/101 (0) bergers disease - piggy 0836544/24/101 (0) Re bergers disease - piggy 085437 4/24/101 (0) http://fourstars.com/board2/board2.html

Bulletin Board Maintained by Independent Executives for: Please post to our new Board. Due to the number of posts here, we have formed a new Bulletin Board to keep load times to a minimum. Be sure to bookmark (add to favorite places) the new board at http://www.fourstars.com/board3/board3.html You can continue to post to board #2 for the next week or so, but please support the move to our new board! Board #3 (June 28 1999 - ?) Board #1 (Dec 1998) Welcome to Fourstars.com bulletin board! This bulletin board is for all Changes Independent Executives, for the purpose of sharing information, ideas and testimonials. Please, no solicitations. Board will be monitored regularly to insure, if ever necessary, the removal of any inappropriate posts. Click Here To Post A Message crohns desease DEAN CARON TURN £6 INTO £6000+ a Work at Home Tracey Work at Home Tracey Re: Work at Home tracey Re: Work at Home tracey DEAD Downlines don't lie.. Successrealm Best MLM pay plan Martin Magraines headaches while pregnant Tammy Romero New Diagnosis Carol What's happening on this board!

29. Re: Bergers Disease Re bergers disease. Follow Ups Post Followup board2 In Reply to Rebergers disease posted by Beverly Baldwin on August 02, 19101 at 142000 http://fourstars.com/board2/messages/1302.html

Re: Bergers Disease Follow Ups Post Followup Posted by Jackie Capman on October 19, 19101 at 22:53:40: In Reply to: Re: Bergers Disease posted by Beverly Baldwin on August 02, 19101 at 14:20:00: : : : Shannon Moore : : : : I have a boyriend who has Bergers Disease and he is now trying Natural Medicine - longer process but might work. He is fit but he has had a relapse after travelling. : : Flaxseed Oil and Nettle Tea is meant to be fantastic. : : Anna : : : : I have just made a friend of a 30+ male who has had the bottom third of his leg amputated...cause? Bergers Disease. I would really like to find out more about this condition, so that I can help him, his wife, and two young kids. : : : : : ,orta by-pass and two big toes amputated. The las top I had was sixteen years ago. Another thing I must mention is that one becomes to fits of giddiness associated with rapid motion. I forgot to mention that I have chronic asthma. : : : : : All of you that are interested, NEVER SMOKE or go into A NIcotine area. : : : : : The origins of the disease are that it originated in Northern France,Brittany, and in Jewish folk.

30. Encyclopedia B-Bm (Search FastHealth.com) Encyclopedia B-Bm skin tumors keratosis Benign stricture of the esophagus Benign Tumors Benzene ingestionBenzodiazepines screen Bereavement bergers disease Beriberi Bernard http://www.jacksonfasthealth.com/encyclopedia/encyclopedia_B-Bm.php

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31. Encyclopedia I-Im (Search FastHealth.com) Encyclopedia I-Im Idiopathic Thrombocytopenic Purpura Idiopathic thrombocytopenic purpura ITP IDMIgA deficiency IgA Nephropathy IgA nephropathy bergers disease IgM mesangial http://www.jacksonfasthealth.com/encyclopedia/encyclopedia_I-Im.php

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32. Titsworth Tree About 1944 he began having trouble with his leggs (bergers disease) andcame back to Texas near Williams area in Brown Co. to farm 14. http://www.flash.net/~barker/ttree.htm

Titsworth Family Tree Descendants of WILLIAM ABRAHANSE TIETSORT Generation No. 1 1. WILLIAM ABRAHANSE1 TIETSORT was born February 08, 1647/48, and died 1722 in DUCHESS COU., NEW YORK. He married NELLY SWART NEELTJE in ULSTER CO., NEW YORK. Child of WILLIAM TIETSORT and NELLY NEELTJE is: 2. i. ISAAC2 TIETSORT, b. 1700; d. November 18, 1757, THE FRONTIER. Generation No. 2 2. ISAAC2 TIETSORT (WILLIAM ABRAHANSE1) was born 1700, and died November 18, 1757 in THE FRONTIER. He married SARA/ZARA LIEUVES/LEWIS, daughter of LEONARD LIEUVES and ELIZABETH HERDENBERG. Child of ISAAC TIETSORT and SARA/ZARA LIEUVES/LEWIS is: 3. i. THOMAS3 TITSWORTH, b. January 04, 1730/31; d. 1819, SULLIVAN CO., KENTUCKY. Generation No. 3 3. THOMAS3 TITSWORTH (ISAAC2 TIETSORT, WILLIAM ABRAHANSE1) was born January 04, 1730/31, and died 1819 in SULLIVAN CO., KENTUCKY. He married ANN TITSWORTH. Child of THOMAS TITSWORTH and ANN TITSWORTH is: 4. i. FREDRICK4 TITSWORTH, b. 1765, WEST VIRGINIA. Generation No. 4 4. FREDRICK4 TITSWORTH (THOMAS3, ISAAC2 TIETSORT, WILLIAM ABRAHANSE1) was born 1765 in WEST VIRGINIA. Child of FREDRICK TITSWORTH is: 5. i. JOHN5 TITSWORTH, b. 1791, LOGAN CO., KENTUCKY; d. June 01, 1872, WASHINGTON CO., ARKANSAS.

33. Ft Communities - MIKE And RAY MENTZER ~ Billk ~ Jul 1, 2001 theaznguy1187 .. ft Popular Status Gender empty Posts 52, hey i found out wassupwith ray, he died in his sleep from kidney failure from bergers disease. http://www.freetrainers.com/FT/jsp/Message.jsp?f_ix=1&m_ix=6021

34. Welcome To The Patient & Family Council's Message Board! My 9 year old was just diagnosed with bergers disease after years of being sick. My23yr old daughter has just been diagnosed with Bergers'disease. http://www.kidney.org/patients/messageboard.cfm

Search Browse Please Select a Topic A to Z Health Guide Bed-Wetting NKF Brochures Online Dialysis Donor Families Health Information High Blood Pressure K/DOQI Kidney Disease Kidney Stones Message Boards and Chats Organ Donation Other Health Topics Transplant Athletics Transplant Recipients Transplantation Urinary Tract Disease Health Care Professionals K/DOQI Recipients Government Relations ... Show Your Support Message Board Hello Everyone: post a message or respond to one. This message board is open to the general public and should not be used as a source for medical information. If you have specific medical questions, you can e-mail them directly to pfc@kidney.org and we will have an appropriate volunteer expert answer your questions. We encourage you to also speak with a health care professional at your treatment center. All you need to do to post a message is to click here . If you do not see your message here after adding it, please hit the reload button ("refresh" on some browsers). Please note that living donation is a beautiful and selfless gift made from the donor to the recipient with no expectations of material compensation. It is illegal to buy or sell human organs. Any comments on the message board that could be construed as allowing money to change hands will be removed immediately. If you would like to be an anonymous donor, contact a local transplant center near you. Sincerely

35. Bergers Glomerulonephritis Of The KidneyBergers Glomerulonephritis Of Kidney Fro A B C DE F G H I J K LM N O P Q R ST U V W X Y Z disease dissyndromesyn A Addison's disease TB histoplasma http://radiology.uchc.edu/eAtlas/GU/1541.htm

36. ESynopsis Of Renal Urinary System Pathology Adult polycystic disease Adult polycystic kidney disease Congenital hydronephrosis Acutepyelonephritis AntiGBM nephritis bergers glomerulonephritis Chronic http://esynopsis.uchc.edu/MGU.htm

37. Synopsis Of Bergers Glomerulonephritis bergers Glomerulonephritis. Pathogenesis • The disease is definedby the presence of IgA immune complexes in the glomerular tufts. http://155.37.5.42/TMGEN/71200043.htm

Bergers Glomerulonephritis (Kidney ) Etiology Probably infectious, especially mucosal infections in respiratory or gastrointestinal tracts. Pathogenesis The disease is defined by the presence of IgA immune complexes in the glomerular tufts. Presumably mucosal derived IgA combines with antigens to form circulating immune complexes that deposit in the glomerular mesangium., Epidemiology This disease is probably the most common form of glomerulonephritis world wide. The disease tends to be mild and relatively asymptomatic so many cases are undiagnosed. Some cases seem to be related to mucosal infections such as respiratory infections and gastrointestinal disorders such as celiac disease and hepatic diseases. Immunogenetics, DR4, ?HLA Bw35. General Gross Description The kidneys in Berger's glomerulonephritis show no gross abnormalities that reflect the pathologic process. General Microscopic Description Berger's disease is a mild form of IgA glomerulonephritis. The glomeruli show modest increase in mesangial cellularity.

38. Diseases Of Kidney polycystic kidney, M, Kidney, Infantile Polycystic Kidney disease. Acute Nephritis.bergers glomerulonephritis, M, Kidney, bergers Glomerulonephritis. http://155.37.5.42/NAV/MSKidney.HTM

Diseases of Kidney Diagnosis G/M Organ Caption Normal glomerulus M Kidney Normal Glomerulus Normal glomerulus M Kidney Normal Glomerular Capillary Loops Normal glomerulus M Kidney Foot Processes Normal Glomerulus M Kidney Normal Glomerulus Normal kidney G Kidney Normal Normal kidney G Kidney Fetal Lobulations Normal kidney G Kidney Normal Kidney Normal kidney M Kidney Normal Adult Kidney Normal kidney M Kidney Normal Adult Kidney Normal kidney M Kidney Normal Adult Kidney Normal kidney M Kidney Normal Fetal Kidney Normal kidney M Kidney Normal Fetal Kidney Normal renal papilla M Kidney Renal Papilla and Calyx Adult polycystic kidney disease G Kidney Adult Polycystic Kidney Disease Adult polycystic kidney disease G Kidney Adult Polycystic Kidney Disease Adult polycystic kidney disease M Kidney Adult Polycystic Kidney Disease Adult polycystic kidney disease M Kidney Adult Polycystic Kidney Disease Adult polycystic kidney disease M Kidney Adult Polycystic Kidney Disease Adult polycystic kidney disease M Kidney Adult Polycystic Kidney Disease Adult polycystic kidney disease G Kidney Adult Polycystic Kidney Disease Congenital hydronephrosis G Kidney Congenital hydronephrosis Congenital hydronephrosis G Kidney Hydronephrosis Congenital hydronephrosis G Kidney Hydroureter and Hydronephrosis Cystic renal dysplasia G Kidney Cystic Renal Dysplasia Cystic renal dysplasia G Kidney Cystic Renal Dysplasia Cystic renal dysplasia G

39. Untitled Document They told me I should see a doctor as soon as possible. The doctorsaid I had a disease called IgA Nephropathy or bergers' disease. http://www.transweb.org/people/recips/lifepost/athletics/us_games/98/people/athp

Luke W. Onken Age: Transplant: Kidney from cadaver donor May 4, 1991 Hometown: Davenport, Iowa Events: 5000 meter, 1500 meter 1500 M race walk, softball throw, three-on-three basketball. My story: My message to the world: Transplants work! Transplants change lives. Transplants save lives. Please consider organ donation. Talk to your family about organ donation. You can be a hero just by signing a donor card. Return to People Index Last modified: 11 May 2000

40. February 2003 - In Cancer Fight, Antiangiogenesis Drugs Still In Play - CAP Toda in tumors, resulting in stabilization and regression of endstage disease. Thissupports the notion that cocktails can improve efficacy, says Dr. bergers. http://www.cap.org/captoday/archive/2003/0203/cancer_symposium_feature.html

February 2003 Feature Story In cancer fight, antiangiogenesis drugs still in play Karen Southwick Researchers at a recent cancer symposium reported encouraging results with several approaches designed to prevent tumors from growing or induce them to destroy themselves. The Dec. 7, 2002 symposium, sponsored by the University of California at San Francisco, singled out anti-angiogenesis drugs as promising and was highlighted by a public appearance by Judah Folkman, MD, the pioneer in such research. Other presentations focused on telomerase inhibitors and viruses designed to stimulate apoptosis in tumors. Although "we can't count on miracle drugs," there should be continued improvement in treatment with new biologic agents, ever-earlier detection, and narrowly targeted therapies, she says. "Incurable cancers will become a chronic disease" that can be kept in check. Dr. Folkman, the Julia Dyckman Andrus professor of pediatric surgery and cell biology at Harvard Medical School, and Gabriele Bergers, PhD, assistant professor in the UCSF Department of Neurosurgery, summarized developments in anti-angiogenesis research, which targets new blood vessels that solid tumors need to grow. The positive regulators of angiogenesis include growth factors such as VEGF, FGF, PDGF, and EGF. Several drugs work against VEGF, including Genentech's Avastin, AstraZeneca's Iressa, and Sugen's SU 11248 and SU 5416. Drugs that block EGF include Iressa, another Genentech drug Tarceva, and ImClone's Erbitux.

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