21. ClinicalTrials.gov - Linking Patients To Medical Research: Browse: C 58. Cerebellar Ataxia (2 studies). 59. cerebellar diseases (4 studies). 60. CerebralArterial Diseases (6 studies). 61. Cerebral Hemorrhage (4 studies). 62. http://www.clinicaltrials.gov/ct/gui/screen/BrowseAny?recruiting=false&path=/bro

22. Department Of Neuroscience At The Johns Hopkins University School Of Medicine: F Psychopathology in Patients With Degenerative cerebellar diseases A Comparisonto Huntington's Disease. Am J Psychiatry. 2002 Aug;159(8)130614. http://neuroscience.jhu.edu/peopledetail.asp?ID=62

23. Neurogate.com Neurosurgery Search Engine And Neurosurgery Custom Medline Searchi Central Nervous System Neoplasms Central Pontine Myelinolysis - CerebellarAtaxia - Cerebellar Cortex - cerebellar diseases - Cerebellar Dyssynergia http://www.neurogate.com/neuro/index2.html

Global Search Add Url Free Medline Contact Us ... Conferences Bookmark this page for your Future Medline/Internet Searches Try the Medline Search Designed for Neurosurgery With FAST results Neurosurgery Internet Search Medline Search Designed for Neurosurgery Enter Keyword to search help Or And Match entire phrase Enter Keyword to search help Categories Premier Neurosurgery Sites General Neurosurgery (MESH 1998 © NLM) Abducens Nerve Abetalipoproteinemia Abnormal Reflex ... Metabolic Brain Diseases General Neurosurgery (MESH 1998 © NLM) Metachromatic Leukodystrophy Microglia Middle Hypothalamus ... Neurogate.com

24. Europe CSC French association for cerebellar diseases. Spain. Switzerland.Association suisse(CH) de l'Ataxie de Friedreich. La présidente http://internaf.org/groups/europe.html

Mainland Europe Europe in general Euro-Ataxia Homepage The international name for this disease is Hereditary Spastic Paraplegia. It is part of the big family of spinocerebellar degenerative hereditary diseases in which the pathologies are somewhat intermediate and hard to differentiate especially in what is called the "complicated" form. However, the "pure" form of S-L is increasingly better known. Many studies are currently undertaken to better understand the main one, SPG4. http://assoc.wanadoo.fr/asl.spastic This spinal, and sometimes cerebellar, degeneration is clinically a part of cerebellar ataxias. That is why in France, we created a federation with the goal to unite all the European organizations (SCD EURO). Concomitantly, a medical network on spinocerebellar degeneration is being created. SCD EURO http://perso.wanadoo.fr/asl.spastic/scdeuro.fr Philippe Grammont 7 D rue des Granges France For A-T For Friedreich's Ataxia and another one CSC : French association for cerebellar diseases.

25. UMHS Department Page Clinical Activities Ataxia clinic. Research Interests cerebellar diseasesBasal ganglia diseases Dementia Positron emission tomography. http://www.med.umich.edu/neuro/resfaculty.htm

Neurology Home Page Neurology Residency Program Educational Philosophy Clinical Training in Neurology Educational Activities Neurology Outpatient Clinics ... Ann Arbor Neurology Faculty This list includes all faculty members who are regularly involved in resident education. The list does not include some faculty members whose primary activity is research, medical student teaching, or clinical practice. SID GILMAN, M.D. William J. Herdman Professor of Neurology Chair, Department of Neurology Director, Michigan Alzheimer's Disease Research Center Clinical Activities: Ataxia clinic Research Interests: Cerebellar diseases Basal ganglia diseases Dementia Positron emission tomography PROFESSORS JAMES W. ALBERS, M.D. Ph.D. Clinical Activities: Director, Neuromuscular clinic

26. Browsing Health Conditions And Diseases C Category Cardiomyopathy Carpal Tunnel Syndrome CatScratch Disease Cataracts Causalgia CeliacDisease Central Pontine Myelinolysis cerebellar diseases Cerebellar Vermis http://www.uksprite.com/search/search/Health/Conditions_and_Diseases/C/

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27. FAMILIAR CEREBELLUM DEGENERATION For a small fraction of cerebellar diseases, there are genetic tests (for example,Freidreich's ataxia, SCA1, SCA-2, and SCA-3). We usually don't order a http://www.medhelp.org/forums/neuro/archive/7551.html

Advertisement Welcome to Med Help International A not-for-profit organization Questions in The Neurology Forum are being answered by doctors from The Cleveland Clinic , consistently ranked one of the best hospitals in America. Subject: FAMILIAR CEREBELLUM DEGENERATION Topic Area: Neurology - General Forum: The Neurology and Neurosurgery Forum Question Posted By: Roy Lieuw Kie Song on Thursday, August 20, 1998 Does any one know where one could go to get help for the above subject? My father has had this for 10 years now, and it is getting worst. The main problem is that he is having problems walking and keeping his balance. Docyors her in Canada have diagnosed him withthe familiar cerebellum degeneration disease and claim that not enough is know about the disease to treat it. As I know that there are ohters who have this, I am wondering if any one is aware of any treatment for this. There are a variety of cerebellar degenerative diseases which run in families. The common symptom produced by damage to the cerebellum (of whatever cause) is ataxia, that is, incoordination. Ataxia can affect gait, causing people to walk like they're drunk (alcohol affects the cerebellum, so that's actually why you walk like that if you drink too much). Ataxia can also affect limb function, so that use of the hands is incoordinated. Familial cerebellar system degenerative diseases can be inherited in dominant or recessive patterns, depending on the specific disease. Dominant is when you have it, a parent has it, and a grandparent has it, as well as assorted brothers, sisters, aunts, uncles, and cousins. Recessive is when two siblings have it but no one else in the family has it.

28. Right Arm Tremor There are cerebellar diseases in which the MRI shows nothing abnormal, but usuallythe whole cerebellum is affected (some biochemical problem, for example) and http://www.medhelp.org/perl6/neuro/archive/14506.html

Advertisement Welcome to Med Help International A not-for-profit organization Questions in The Neurology Forum are being answered by doctors from The Cleveland Clinic , consistently ranked one of the best hospitals in America. Subject: Right Arm Tremor Topic Area: Movement Forum: The Neurology and Neurosurgery Forum Question Posted By: sally on Sunday, December 20, 1998 Dear Doctor, Let me type our findings on the MRI. This is just the impression. There is probably dilated perivesicular space at the inferior aspect of the basal ganglia on the right side. There is slight prominence of the cisterna magna. No mass lesion is seen. No abnormal enhanced lesion is identified with gadolinium injection. There is minimal mucasol thickening demonstraed in the max. sinuses. His right arm tremor is still with him sometimes it seems worse than others, but he is on vacation from school so he hasnt been writing as much. Although he did say that when he was trying to drink the other night he did have some difficulty. We will see his Doctor on Tuesday to get the blood results of a thyroid and test for Wilsons Disease. but I still would appreciate any suggestions you might have. I truely appreciate how quickly you returned my last posted note thank you again and have a Happy New Year!!! Sally The MRI interpretation suggests that there is no structural damage to the cerebellum or the connections with other parts of the brain. If the problem is in the cerebellum, AND if it is localized to one side or one limb, then you would expect some abnormality. There are cerebellar diseases in which the MRI shows nothing abnormal, but usually the whole cerebellum is affected (some biochemical problem, for example) and not just the part that controls one limb. Therefore, I suspect the cerebellum is not involved (based on limited data, of course, in this forum).

29. Open Directory - Health: Conditions And Diseases: C Causalgia@ (6); Celiac Disease@ (43); Central Pontine Myelinolysis@ (3);cerebellar diseases@ (4); Cerebellar Vermis Agenesis@ (5); Cerebral http://www.mptdo.com/Health/Conditions_and_Diseases/C/

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30. NINDS News Articles - Current The study suggests that patients with cerebellar diseases may benefit from screeningand treatment of psychiatric symptoms. Full Article. News article archive. http://accessible.ninds.nih.gov/news_and_events/news_articles_index.htm

News and Events - National Institute of Neurological Disorders and Stroke (NINDS) Skip menus Main sections of the NINDS web site Home About NINDS Disorders ... Find People News and Events section pages and search Image Description Science For the Brain The nation's leading supporter of biomedical research on disorders of the brain and nervous system. Select Topic Disorder Quick Links Alzheimer's Autism Cerebral Palsy Chronic Pain Epilepsy Headache Multiple Sclerosis Parkinson's Stroke Traumatic Brain Injury Press releases Current - you are in this section Archived Events Proceedings NINDS Notes News articles Current - you are in this section Archived Online events Upcoming Archived Of interest ... Labs at NINDS NINDS Search (search help) Contact us My privacy NINDS is part of the National Institutes of Health Contact us Content for this page NINDS News Articles - Current A New Test for Myotonic Dystrophy: Exposing an Enemy That’s Too Big to See For release: Wednesday, March 26, 2003 Researchers have developed a genetic test that detects a common form of muscular dystrophy with 99 percent accuracy. The accurate diagnosis of myotonic muscular dystrophy type 2 (DM2) allows researchers to fully describe its clinical features for the first time. Full Article Fact Sheet Dystonia Protein Linked to Problem Common in Other Neurological Disorders For release: Monday, March 24, 2003

31. FA FAQ Eds. Vinken, Bruyn, and Klawans (Elsevier and Amsterdam, New York, 1992)3. The Handbook of cerebellar diseases, Ed. Richard Lechtenberg http://www.fortnet.org/fapg/faq.htm

Friedreich's Ataxia - Frequently Asked Questions What is Friedreich's Ataxia? What are the major symptoms of FA? What other symptoms might occur? How is FA passed on in the family? ... What should somebody with FA do? What is Friedreich's ataxia? Friedreich's ataxia (FA) is a very rare neurological genetic disorder, that affects speech, balance and coordination. In some respects it is similar to multiple sclerosis. It is an inherited genetic disease in which a person is born with. Most people who have Friedreich's Ataxia, eventually need support to walk, like a scooter, walker or wheelchair. Although, not deemed terminal, it is progressive and as a result, complications can occur to the respiratory systems, (heart and lungs etc.) Friedreich's ataxia was the first form of hereditary ataxia to be distinguished from other forms of ataxia. It is named after the German doctor, Nikolaus Friedreich, who first described it in 1863. Presently there is no known cure, but please don't fret or become discouraged, because with the advancement of all the recent genetic discoveries and research, there is great deal of hope indeed! What are the major symptoms of Friedreich's ataxia?

32. Psychology Arena - Subject Listing Of Psychology Journals Title cerebellar diseases. Author(s) Holly A. Shill; Mark Hallett.Journal International Review of Psychiatry. Year 2001 Volume http://gessler.ingentaselect.com/vl=1/cl=7/ini=psyarena/nw=1/rpsv/catchword/carf

Document Request Title: Cerebellar diseases Author(s): Holly A. Shill ; Mark Hallett Source: International Review of Psychiatry Volume: Number: Page: DOI: Publisher: Abstract: Cerebellar diseases are recognized by a combination of clinical findings consisting of gait ataxia, dysarthria, incoordination and eye movement abnormalities. The patient with a cerebellar syndrome can be classified by age of onset of symptoms and course of illness. In general, an acute presentation is more likely to have a symptomatic cause whereas a chronic, progressive course is likely to represent a neurodegenerative condition. Increasingly, the spectrum of cerebellar degeneration is being elucidated as molecular techniques identify genetic causes. This review synthesizes the clinical features of cerebellar dysfunction and presents a method for classifying the cerebellar disorders. Evaluation would then follow according to this classification and prognosis and treatment based on specific etiology determined. Reference Links: The requested document is freely available only to registered users with an online subscription to International Review of Psychiatry You can purchase this article below Click here for the publisher's subscription information This journal is hosted by Ingenta Select, who have not recognised you as a registered user.

33. HMS Neurobiology-Yellen the role of BDNF in normal cerebellar development, we have also demonstratedthat neurotrophins, in particular NT3, have a role in cerebellar diseases. http://neuro.med.harvard.edu/site/faculty/segal.html

Selected Publications: Return to list of Faculty Rosalind Segal, M.D., Ph.D. Segal Lab Associate Professor of Neurobiology Harvard Medical School Department of Pediatric Oncology Dana-Farber Cancer Institute Our lab is interested in mechanisms whereby extracellular stimuli regulate proliferation and survival in the developing nervous system. Neurotrophin Signaling In order for target-derived neurotrophins to regulate the survival of a developing presynaptic cell, a signal must be propagated from the nerve terminal along the axon to the nucleus. We found that activated Trk receptors function as rapid retrograde signal carriers to elicit neuclear responses to target derived neurotrophins. Our data indicate that the mechanism of signal propagation is retrograde vesicular transport of activated Trk-ligand complexes. Once they reach the cell body, activated receptors elicit nuclear responses-including

34. Funded Research - MRC Nervous System/enzymology; Central Nervous System/physiology; Centrifugation;Centromere; cerebellar diseases; Cerebellum; Cerebral Aneurysm; http://fundedresearch.cos.com/cgi-bin/MRC/keyword/main?letter=C

35. MITECS: Cerebellum Early in the twentieth century, neurologists defined the unique symptoms,such as dysmetria and motor incoordination, of cerebellar diseases. http://cognet.mit.edu/MITECS/Articles/ito

Cerebellum The cerebellum constitutes 10 to 15 percent of the entire brain weight, about 140 grams in humans. Rollando (1809; see Dow and Moruzzi 1958) was the first who, by observing motor disturbances in an animal with a lesioned cerebellum, related the cerebellum to movement. Careful analyses of the motor disturbances so induced led Flourens (1824; see Dow and Moruzzi 1958) to conclude that the cerebellum is neither an initiator nor an actuator, but instead serves as a coordinator of movements. An animal with a damaged cerebellum still initiates and executes movement, but only in a clumsy manner. Flourens (1842) and Luciani (1891; see Dow and Moruzzi 1958) observed that motor disturbances caused in an animal by a partial lesion of the cerebellum were gradually compensated for due to the functional plasticity of cerebellar tissues. According to the current knowledge cited below, this plasticity is an expression of a learning capability of the cerebellum, which normally plays a role in MOTOR LEARNING , as in the cases of practicing sports and acquiring skilled movements. Early in the twentieth century, neurologists defined the unique symptoms, such as dysmetria and motor incoordination, of cerebellar diseases. Based on these classic observations, it has been thought that the major function of the cerebellum is to enable us to learn to perform movements accurately and smoothly. The extensive studies that have been performed over the past four decades have facilitated the formulation of comprehensive views on the structure of the cerebellum, what processes occur there, and what roles it plays not only in bodily but also in cognitive functions, even though some of the views are still hypothetical.

36. Searchalot Directory For C Causalgia (6); Celiac Disease (42); Central Pontine Myelinolysis (3);cerebellar diseases (4); Cerebellar Vermis Agenesis (5); Cerebral http://www.searchalot.com/Top/Health/ConditionsandDiseases/C/

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37. Pages.infinit.net/macmike/internaf/archives/FA.txt Vinken, Bruyn, and Klawans (Elsevier and Amsterdam, New York, 1992)3. The Handbook of cerebellar diseases, Ed. Richard Lechtenberg http://pages.infinit.net/macmike/internaf/archives/FA.txt

38. R&D: Medicine - Neurology psychophysics (perception of motion, proprioceptive perception) Kinematic investigations(Elite system) in various cerebellar diseases Investigation of the http://www.uni-essen.de/fet&ww/fue/eng/fb14/neurolo.htm

University of Essen Medicine, University Hospital, Medical School Prof. Dr. H. C. Diener Neurology Research Profile: Contact: Prof. Dr. med. H. C. Diener phone: 0201/723-2461 fax: 0201/723-5901 Priv.-Doz. Dr. med. C. Zimmermann phone: 0201/723-2364 fax: 0201/723-5901 Imaging Quantitative determination and therapy of incipient arteriosclerotic alterations in the human carotid arteries using 3-D reconstructions of images of the carotid artery Histological morphology of sonographic patterns in duplex sonography Dynamic and static models of brain glucose metabolism using FDG and PET. Stroke research A TIA model in rats using electroretinography during and after carotid occlusion Functional reorganization of the brain after stroke; RCBF measurements using OCO inhalation and PET Effect of brain selective Ca antagonists on recent strokes. Neuroimmunology Animal experiments on the acute inflammatory meningeal reaction at the blood/brain barrier and on meningeosis carcinomatosa Investigations of natural and pathological autoimmunity in neuromuscular diseases using the techniques of analytical and preparative biochemistry Neuroimmunological questions which are being primarily investigated using the model of murine toxoplasma encephalitis and listeriosis.

39. Institut Für Physiologie - Abteilungen Translate this page Genetic defects and phenotypical outcome in cerebellar diseases. Functional neuroanatomyof the peripheral vestibular system. Publikationen Seit 1992. http://www.fu-berlin.de/physiol/Departments.html

Institut für Physiologie - Abteilungen Korrekturen und Änderungswünsche bitte an webmaster Physiologie Neurophysiologie Prof. Dr. Siegel PD Dr. Bäurle Vegetative Physiologie ... Prof. Dr. Gaehtgens (beurlaubt: Präsident der FU-Berlin) Prof. Dr. Pries Prof. Dr. Habazettl S-Professur: "Physiologie und Pathophysiologie der Organperfusion" (DHZB) Prof. Dr. Kübler S-Professur: "Physiologie und Pathophysiologie vaskulärer Regulationen" (DHZB) Prof. Dr. Kirsch Gäste und weitere Mitglieder des Lehrkörpers Einrichtungen des Institutes ... Prof. Dr. A. Pries Tel (+49 30) 8445 1632 Sekretariat Telefon Fax Frau A. Scheuermann Labor Telefon Fax Frau S. May Mitarbeiter: Dr. Mauro Bongrazio Dr. Luis Da Silva-Azevedo Dipl. med. Bettina Reglin Dr. Annemiek Cornelissen ... Dipl.Ing. Jürgen Palow Forschungsgebiete: Rheologie des Blutes.

40. Indexing Initiative Prototype Cognition (12). Globus Pallidus (13). Basal Ganglia (14). Motor Activity (15).cerebellar diseases (16). Frontal Lobe (17). Mental Processes (18). Brain (19). http://ii.nlm.nih.gov/Demo/Citations/98017457.html

link to PubMed: Medline Citation: MEDLINE indexed MeSH expressions are displayed in the left column. MeSH expressions that concur with terms suggested by the Indexing Initiative prototype are displayed in red. The number in parenthesis indicates the prototype's rank order. out of starred MeSH expressions are suggested by Indexing Initiative prototype. out of starred MeSH expressions are suggested by Indexing Initiative prototype. Indexed MeSH Expressions Additional Suggested MeSH Expressions *Brain Mapping (7) *Cerebellum (1) *Motor Cortex (2) Animal (check tag) Human (check tag) Magnetic Resonance Imaging Neural Pathways (6) Neurons (11) Prefrontal Cortex (8) Primates Thalamus (3) Cerebral Cortex (4) Cerebellar Nuclei (5) Cerebellar Cortex (9) Movement (10) Cognition (12) Globus Pallidus (13) Basal Ganglia (14) Motor Activity (15) Cerebellar Diseases (16) Frontal Lobe (17) Mental Processes (18) Brain (19) Cerebellar Neoplasms (20) Macaca (21) Haplorhini (22) Thalamic Nuclei (23) Red Nucleus (24) Macaca nemestrina (25)

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