41. Answer List 1, 1996 Tumor L Lactic Dehydrogenase Laparoscopy Legionella Pneumophila Leiomyoma LeiomyosarcomaLeukotrienes Lichen Sclerosus lipoid nephrosis Lupus Nephritis Lymph http://www.kumc.edu/instruction/medicine/pathology/ed/exams/ans1_96.html

Answer List #1, 1996 Click on a circle to insert into currently selected question A- Abrasion Abscess Acid Phosphatase Acute Interstitial Nephritis Acute Tubular Necrosis Adenovirus Adult Polycystic Kidney Disease Adult Respiratory Distress Syndrome (ARDS) Alkaline Phosphatase Allergic Acute Interstitial Nephritis Allergic Bronchopulmonary Aspergillosis Alpha-1-Antitrypsin Alpha Fetoprotein Alport Syndrome Amyloidosis Analgesic Nephropathy Anorchia Anthracosis Anuria ARDS Artery Arthritis Asbestos Asbestos Body Asbestosis Aspergillosis Aspiration Of Gastric Contents Asthma: Allergic Asthma: Infectious Asthma: Occupational Asthma: Exercise Induced Asthma: Drug Induced Atelectasis Autosomal Dominant Polycystic Kidney Autosomal Recessive Polycystic Kidney Azoospermia B- Bacteremia Benign Familial Hematuria Berger's Disease Bilateral Cortical Necrosis Bladder, Urinary Blastomycosis Bone Bordetella Pertussis Bowen Disease Bowman Capsule Bronchiectasis Bronchiole Bronchiolitis Obliterans-Organizing Pneumonia (B00P) Bronchitis: Acute Bronchitis: Chronic Bronchogenic Cyst Bronchopleural Fistula Bronchopneumonia Bronchoscopy C- Calcium Carbonate Calcium Chloride Calcium Hydroxyapatite Calcium Oxalate/Phosphate Calculus Carcinoid Carcinoma: Adenocarcinoma Carcinoma: Bronchioloalveolar Carcinoma: Carcinoma In Situ Carcinoma: Embryonal Carcinoma Carcinoma: Endometrioid Carcinoma Carcinoma: Infiltrating Duct Carcinoma Carcinoma: Lobular Carcinoma: Medullary Carcinoma: Mucinous Carcinoma: Oat Cell Carcinoma Carcinoma: Prostatic Carcinoma: Renal Cell

42. Directory :: Look.com Disabilities (196) Leishmaniasis (21) Leprosy (15) LeschNyhan Syndrome (5) Leukemia(98) Leukodystrophy (31) Lice (20), lipoid nephrosis (5) Lissencephaly (5 http://www.look.com/searchroute/directorysearch.asp?p=43354

43. Searchalot Directory For L 21); Leprosy (15); LeschNyhan Syndrome (5); Leukemia (98); Leukodystrophy(31); Lice (20); lipoid nephrosis (5). Lissencephaly (5); Liver http://www.searchalot.com/Top/Health/ConditionsandDiseases/L/

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44. Nephrology Terms entry LifeSite™ hemodialysis venous access system see MT Desk glossary entryline of Toldt lipoid nephrosis minimal-change disease lithium Lithospec http://mtdesk.com/lstneph.shtml

Medical transcription and terminology site offers weekly updates of new medical and surgical terms, medical dictionary, A-Z index of new terminology, word lists, sample operative reports, links to the best medical information on the web by topic, public forums, free classified ads, and live chat. Discounts up to 90% off - New 2002 References are HERE! Monday, March 31, 2003 NEPHROLOGY TERMS Back to MT DESK Back to Index of Terminology A-M N-Z Back to Word List Index Nephrology Terms Tc-DMSA Tc-DTPA Tc-labeled HAG3 [investigational imaging agent] Tc-labeled MAG3 Tc-MAG3 access blood flow access flow measurement access recirculation (AR) ACE (angiotensin-converting enzyme) ACE inhibitor ACEI (angiotensin-converting enzyme inhibitor) acid-base homeostasis acidification acquired cystic disease [dialysis-associated cystic disease] acquired renal insufficiency Acucise® endopyelotomy [balloon] catheter - Applied Medical Resources Acucise endopyelotomy acute nephritic syndrome acute poststreptococcal glomerulonephritis (ASPGN) acute renal failure (ARF) acute tubular necrosis (ATN) ADPKD (autosomal dominant polycystic kidney disease) adrenal gland adult-onset medullary cystic disease aerobic bacteria air embolism Aksys PHD™ system [investigational hemodialysis system for home use] - see MT Desk glossary entry Alcaligenes xylosoxidans ALG (antilymphocyte globulin) Allen-Brown shunt allergic interstitial nephritis allogenic renal transplant allograft dysfunction AlloMune™ system - see MT Desk glossary entry allopurinol (Zyloprim®)

45. Nefrología. Volumen 4/99 Casos Clínicos. Art.2 Translate this page Kidney Int 33 708-715, 1998. ·4.·Kashgarian M lipoid nephrosis and focal sclerosisdistinct entities or spectrum of disease. Nephron 13 105-108, 1974. http://www.aulamedica.es/nefrologia/nefro2-00/comunic1e.htm

Volumen XX. Número 2. 2000 INDICE Factores de pronóstico en la evolución de la glomeruloesclerosis segmentaria y focal D. Moreno*, Ch. Magrans**, H. Gómez Barry*** y A. M. Gámez**** *Especialista de Segundo Grado en Nefrología. Jefe del Servicio de Trasplantes del Instituto de Nefrología. **Profesor de Nefrología del Instituto de Nefrología. ***Profesor de Patología. Jefe del Departamento de Patología del Instituto de Nefrología. ****Nefróloga Hospital Julio Trigo. Instituto de Nefrología "Abelardo Puch López". La Habana. Cuba. Recibido: 14-VI-99. En versión definitiva: 28-XII-99. Aceptado: 28-XII-99. Correspondencia: Dr. Darío Moreno Vega Servicio de Trasplantes Instituto de Nefrología «Abelardo Buch López» Ave. 26 y Rancho Boyeros Plaza Habana, 6 Cuba Introducción Resultados Subir Las nefropatías glomerulares primarias ocupan un lugar preponderante en el campo de la Nefrología, ya que constituyen una de las primeras causas de insuficiencia renal crónica . Aunque la etiología de este conjunto de enfermedades no está totalmente dilucidada en el momento actual, su patogenia, aun siendo compleja, está mejor conocida, ya que a partir de los trabajos de Brenner han emergido un conjunto de conocimientos sobre los mecanismos de lesión glomerular, desencadenados a partir de estímulos, sean éstos de carácter inmunológico o de otra índole, que conllevan en última instancia la pérdida de unidades funcionales

46. MEDLINEplus Medical Encyclopedia: Minimal Change Disease Minimal change disease is defined, along with a list of alternate names, causes, incidence and risk factors.Category Health Conditions and Diseases Glomerular Minimal Change...... Alternative names Return to top Minimal change nephrotic syndrome; Nil disease;lipoid nephrosis; Idiopathic nephrotic syndrome of childhood. http://www.nlm.nih.gov/medlineplus/ency/article/000496.htm

Skip navigation Medical Encyclopedia Other encyclopedia topics: A-Ag Ah-Ap Aq-Az B-Bk ... Z Minimal change disease Contents of this page: Illustrations Alternative names Definition Causes, incidence, and risk factors ... Prevention Illustrations Male urinary system Alternative names Return to top Minimal change nephrotic syndrome; Nil disease; Lipoid nephrosis; Idiopathic nephrotic syndrome of childhood Definition Return to top A disorder of the kidneys that affects the structures (glomeruli) which include small capillaries surrounded by membranes through which the blood is filtered to form urine. Causes, incidence, and risk factors Return to top Minimal change disease is one cause of nephrotic syndrome . It is named because under a light microscope the glomeruli appear totally normal. Under an electron microscope characteristic changes in the glomeruli can be seen, including the fusion of a portion of the epithelial layer. Minimal change disease is most common in children but occasionally occurs in adults. The cause is unknown, but the disease may be preceded by viral infection, allergic reactions or recent immunizations. Minimal change disease does not reduce the amount of urine produced. It rarely progresses to

47. MEDLINEplus Medical Encyclopedia: Topics Beginning With L-Ln risk profile; Lipofuscin; Lipofuscinosis; lipoid nephrosis see Minimalchange disease. Lipoprotein test see Cholesterol test; Lipoprotein http://www.nlm.nih.gov/medlineplus/ency/encyclopedia_L-Ln.htm

Skip navigation Other encyclopedia topics: A-Ag Ah-Ap Aq-Az B-Bk ... Z Medical Encyclopedia Topics beginning with "L-Ln" Labyrinthitis Laceration see Wounds Lack of coordination see Movement - uncoordinated Lack of intrinsic factor Lack of menses see Menstruation - absent Lack of regular bowel movements see Constipation Lack of sensation see Numbness and tingling Lack of strength see Weakness Lack of thirst see Thirst - absent Lacquer Lacrimal gland tumor Lactase deficiency see Lactose intolerance Lactate see Lactic acid Lactate dehydrogenase see LDH Lactate dehydrogenase isoenzymes see LDH isoenzymes Lactation see Breastfeeding Lactation - abnormal see Nipple discharge - abnormal Lactic acid Lactic acid dehydrogenase see LDH Lactic acid overdose Lactic acidosis Lactic dehydrogenase isoenzymes see LDH isoenzymes Lacto-ovovegetarian see Vegetarianism Lactose intolerance Lactose tolerance test Lacto-vegetarian see Vegetarianism Laennec's cirrhosis see Alcoholic liver disease Lambert-Eaton Syndrome Lamellar ichthyosis Laminagraphy see Chest tomogram Lancefield group B streptococcus see Group B streptococcal septicemia of the newborn Landry-Guillain-Barre syndrome see Guillain-Barre syndrome Langerhans cell histiocytosis see Histiocytosis Language disorder - expressive see Expressive language disorder - developmental Language impairment see Speech impairment Lanolin overdose Lanoxin, Purgoxin, Allocar see

48. Minimal Change Glomerulopathy From the Renal Pathology Tutorial an article beginning with all the synonyms for this disease.Category Health Conditions and Diseases Glomerular Minimal Change...... Minimal Change Glomerulopathy. There are many synonyms for minimal change glomerulopathy,eg, minimal change disease, lipoid nephrosis, nill disease. http://www.gamewood.net/rnet/renalpath/ch4.htm

Renal Pathology Tutorial Written by: J. Charles Jennette, MD Produced by: F.W. Maddux, MD Renal Pathology Tutorial Home Page Normal Histology Clinical Presentation ... Type II MPGN Minimal Change Glomerulopathy Slide 12 shows the characteristic light microscopic finding, i.e., no abnormality. Sometimes there may be a little bit of mesangial hypercellularity in a few segments. Otherwise, any scarring, any infiltration of leukocytes, any necrosis, or any other substantial structural changes in glomeruli rule out a diagnosis of minimal change glomerulopathy. Slide 13 The ultrastructural finding diagramed in Slide 14 are effacement of visceral epithelial foot processes and epithelial microvillous transformation. Microvillous transformation of epithelial cytoplasm often accompanies effacement. The effacement of foot processes and microvillous transformation are not specific for minimal change glomerulopathy. Foot process effacement is characteristic for minimal change glomerulopathy and is required for the pathologic diagnosis of this disease; however, this same change is present in any patient with substantial proteinuria of any cause. Therefore, the diagnosis of minimal change glomerulopathy is one of exclusion, i.e., these ultrastructural changes should be present in the absence of light microscopic, immunohistologic or other ultrastructural features of any other cause of proteinuria. The electron micrograph in Slide 15 is from a patient with minimal change glomerulopathy and shows almost complete effacement of the visceral epithelial foot processes. There is condensation of the epithelial cytoskeleton near the basement membrane. If you don't know what this is, you can mistake it for subepithelial electron dense deposits, suggesting membranous glomerulopathy. It is actin condensation that takes place inside of visceral epithelial cytoplasm when there is effacement of foot processes, suggesting that there is movement of cytoplasmic structures during the effacement event.

49. Case Of The Month type I K. Membranoproliferative glomerulonephritis, type II L. Membranous nephropathyM. Minimal change disease (lipoid nephrosis) N. Nodular or diffuse http://www.amsa.org/tnp/com/com12_01.cfm

THE NEW PHYSICIAN Search AMSA: AMSA Home Become a Member Site Index Contact Us Mar 31, 2003 Site Directory About AMSA Medical Education Health Policy Global Health ... Resources case of the month December 2001, Volume 50, Number 9 What's the Diagnosis? Concerning the kidney Case Studies: Match each numbered statement with the most appropriate lettered item. Each lettered item can be used once, more than once or not at all. A. Acute glomerulonephritis B. Alport syndrome C. Amyloidosis D. Chronic glomerulonephritis E. Crescentic glomerulonephritis F. Diffuse proliferative glomerulonephritis G. Focal proliferative glomerulonephritis H. Focal necrotizing glomerulonephritis I. Focal segmental glomerulosclerosis J. Membranoproliferative glomerulonephritis, type I K. Membranoproliferative glomerulonephritis, type II L. Membranous nephropathy M. Minimal change disease (lipoid nephrosis) N. Nodular or diffuse diabetic glomerulosclerosis Oliguria, proteinuria, hematuria and generalized edema developed in a 6-year-old boy two weeks after a severe throat infection, after which a renal biopsy was performed. The glomeruli showed hypercellularity, reflecting endothelial and mesangial cell proliferation and infiltrates of neutrophils and macrophages. Mild proteinuria and microscopic hematuria were found in a 25-year-old man. In the renal biopsy, some glomeruli showed mesangial proliferation, with mesangial deposits of IgA. The clinician told the patient that this is the most common form of glomerular disease and that in most cases it has a good prognosis. No treatment was given.

50. GI, Metabolism & Nutrition Module 1000 1200, Glomerular Diseases - Nephrotic Syndrome - lipoid nephrosis,Focal Segmental Glomerulosclerosis, MembranoL s Nephropathy, Diabetic http://www.smbs.buffalo.edu/CTF2/renal.htm

Curriculum Blocks Year 1 Year 2 Year 3 Year 4 Renal Module Instructors Books Appendices Schedule INSTRUCTORS J.M. Goldinger, Ph.D. Professor of Physiology, Course Director B.K. Stefanick, Ph.D. Assistant Prof. of Pathology JW Lohr, M.D. Associate Prof. of Medicine and Pharmacology JR Cotter, M.D. Associate Prof. of Anatomical Science A.E. Brownie, Ph.D. Distinguished Teaching Professor of Biochemistry B.M. Murray, Ph.D. Associate Prof. of Medicine M.W. Stinson, M.D. Professor of Microbiology R.C. Venuto, Ph.D. Professor of Medicine J.E. Springate, M.D. Associate Prof. of Pediatrics B.K. Mookedee, M.D. Professor of Medicine A.J. Lesse, M.D.,Ph.D. Associate Prof. of Pharmacology R.R. Heffner, M.D.

51. Www.nber.org/mortality/1995/docs/ch10.txt glomerulonephritis mixed membranous and proliferative glomerulonephritis 581.3 Withlesion of minimal change glomerulonephritis lipoid nephrosis Minimal change http://www.nber.org/mortality/1995/docs/ch10.txt

52. L Leukemia; @ Leukodystrophy; @ Lice; @ lipoid nephrosis; @ Lissencephaly;@ Liver Disorders; @ Locomotor Ataxia; @ Lowe Syndrome. @ Lumbar Disk; http://www.ad.com/Health/Conditions_and_Diseases/L/

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53. Conventional Therapy (MCGN, minimal proliferative intercapillary glomerulonephritis with nephrotic syndrome(MPI + NS), glomerular minimal changes, lipoid nephrosis, minimal change http://www.uni-tuebingen.de/uni/kmp/cgts5e.htm

Next Back Home Conventional Therapy Patient Recruitment Stopped Primary Glomerulonephritis General information Objective Conventional therapy protocols are assigned to a patient by random numbers in the case of several available treatment modalities for one and the same form of glomerulonephritis. The treatment effect is compared by a randomized, controlled, open trial. The randomization is done on-site at each centre by a computer program (random number generator). Inclusion criteria: The inclusion criteria for the treatment following the underneath mentioned therapy protocol are: For all glomerular diseases except IgA-Nephropathy: 1. Proteinuria over 3.5 g/die, measured three times in series IgA-Nephropathy: Histologically proven diagnosis independently of serum creatinine or proteinuria. If inclusion criteria are not given and the patient is suffering from biopsy proven glomerulonephritis patients data and follow-up are nevertheless registered in the central data bank. Analysis on this subgroup of patients will be performed separately. Study endpoint The study endpoint is determined five years after inclusion. An intermittent analysis of the data will be done after six months.

54. Biblioteca Virtual En Salud Onchocerca volvulus also leads to membranoproliferative glomerulonephritis andlipoid nephrosis. Renal involvement with Wuchereria bancrofti is rare. http://bvs.insp.mx/componen/svirtual/ppriori/nbibliodet.asp?idb=5129

55. Liposyn Overdose Liposyn Contraindications Liposyn II - RxList Monographs Fat Emulsion) is contraindicated in patients demonstrating disturbances in normalfat metabolism such as pathologic hyperlipemia, lipoid nephrosis or acute http://www.rxlist.com/cgi/generic3/liposyn_od.htm

Liposyn II Health News Please, take our 1 second survey! SEASONAL DEPRESSION MENTAL HEALTH ... WEIGHT LOSS OVERDOSE In the event of fat overload during therapy, stop the infusion of Liposyn II (Intravenous Fat Emulsion) until visual inspection of the plasma, determination of triglyceride concentrations, or measurement of plasma light-scattering activity by nephelometry indicates the lipid has cleared. Re-evaluate the patient and institute appropriate corrective measures. See WARNINGS and PRECAUTIONS CONTRAINDICATIONS The administration of Liposyn II (Intravenous Fat Emulsion) is contraindicated in patients demonstrating disturbances in normal fat metabolism such as pathologic hyperlipemia, lipoid nephrosis or acute pancreatitis if accompanied by hyperlipemia. With the exception of heparin at 1 to 2 units/mL of fat emulsion, additives to the Liposyn II bottle are contraindicated. Partly used containers must not be stored for later use. Filters must not be used with Liposyn II. Do not use any bottle in which there appears to be an oiling out of the emulsion. This Page Last Revised 12/31/02

56. Browsing Health Conditions And Diseases L Category Syndrome Learning Disabilities Leishmaniasis Leprosy LeschNyhan Syndrome, LeukemiaLeukodystrophy Lewy Body Disease Lice lipoid nephrosis Lissencephaly Liver http://www.uksprite.com/search/search/Health/Conditions_and_Diseases/L/

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57. Carlos J. Rodriguez-Feo, DDS, Oral And Maxillofacial Surgery - About The Doctor of Arts and Sciences in Washington, DC, completing research in electron microscopyon an animal model of the childhood kidney disease, lipoid nephrosis. http://www.jawsurgery.org/aboutus.html

Home Map and Directions About the Doctor Dental Implant Information Impacted Teeth Contact Us About the Doctor Carlos J. Rodriguez-Feo, DDS, Oral and Maxillofacial Surgery Comprehensive adult and pediatric oral and maxillofacial surgery. What we do Services provided encompass a full scope of pediatric and adult oral and maxillofacial surgery. This includes office anesthesia, dentoalveolar surgery, and surgical correction of maxillofacial skeletal deformities, maxillofacial trauma surgery, temporomandibular joint (TMJ) surgery, surgical management of pathological conditions and reconstructive surgery Our business history Dr. Carlos J. Rodriguez-Feo was born in Habana Cuba. Raised in Detroit, Michigan during his early childhood, he later moved to Miami, Florida. He graduated salutatorian in 1980 from Christopher Columbus High School in Miami. In 1983, he earned a Bachelor of Sciences in Biology, Magna Cum Laude, at Georgetown University College of Arts and Sciences in Washington, D.C., completing research in electron microscopy on an animal model of the childhood kidney disease, lipoid nephrosis. Continuing in his studies in Washington, he earned a Doctor of Dental Surgery, Summa Cum Laude, in 1987 from Georgetown University School of Dentistry. He passed the National Board Dental Examinations, averaging in the 99th and 98th percentile. In 1987, he qualified for and received his license from the Florida State Board of Dentistry. Continuing his education with advanced specialty training at Georgetown University, he completed a residency in Oral and Maxillofacial Surgery in 1990. He received average scores in the 98th and 97th percentile on the Oral and Maxillofacial Surgery Intraining Examinations. During residency he worked at Georgetown University Medical Center, the National Institutes of Health (NIH), DC General Hospital and the Veterans Administration Medical Center in Washington, D.C. He served as Chief Resident at each institution during 1989 to 1990. While on the Junior Active Staff of the Clinical Center of NIH, he participated in various research projects, including studies in the management of surgical stress and acute pain, in evaluation of triazolam, nitrous oxide and diazepam in oral surgery, in inflammatory mediators, in acute pain and pain control, in dental implants and in techniques of rigid fixation in orthognathic surgery.

58. SpringerLink: Pediatric Nephrology - Table Of Contents Vol. 12 Issue 5 Paul Laflam, Lamar Chandler experimental studies, original article Antiinterleukin8 antibody abolishes effects of lipoid nephrosis cytokine Pediatr Nephrol http://link.springer-ny.com/link/service/journals/00467/tocs/t8012005.htm

Pediatric Nephrology ISSN: 0931-041X (printed version) ISSN: 1432-198X (electronic version) Table of Contents Vol. 12 Issue 5 transplantation, original article : Weight-/height-related bone mineral density is not reduced after renal transplantation Pediatr Nephrol Article in pdf format (154 KB) LuAnn Klemme, Alfred J. Fish, Stephen Rich, Beryl Greenberg, Beverly Senske, Miriam Segall: genetic renal disease, original article : Familial ureteral abnormalities syndrome: genomic mapping, clinical findings Pediatr Nephrol Article in pdf format (416 KB) Mary B. McBride, Sue Rigden, George B. Haycock, Neil Dalton, William Van't Hoff, Lesley Rees, G. Venkat Raman, Fernando Moro, Chisholm S. Ogg, J. Stewart Cameron, H. Anne Simmonds: genetic renal disease, original article : Presymptomatic detection of familial juvenile hyperuricaemic nephropathy in children Pediatr Nephrol Article in pdf format (197 KB) Ewa Elenberg, Laura L. Norling, Ronald E. Kleinman, Julie R. Ingelfinger: genetic renal disease, original article : Feeding problems in cystinosis Pediatr Nephrol Article in pdf format (133 KB) Lawrence R. Shoemaker, William Bergstrom, Kevin Ragosta, Thomas R. Welch:

59. Medical Library Online. Journals, Medical Reference Books, February 5, 2003 Lipodystrophy, Acquired Partial. Lipodystrophy, Centrifugalis Abdominalis Infantilis.lipoid nephrosis. Lipoid Proteinosis. Lipomas. Lipomastia. Liposuction. http://www.medical-library.org/journals2a/med_library_disease_l.htm

This page has moved. Click here to view. Medical Library. Full Text Journals Medical Books Online. Physicians Drug Reference L *Labetalol Large granular lymphocytes (LGL) Large granular lymphocytic (LGL) leukemia Legionella pneumophila Legionnaire's disease Leptospirosis (Leptospira) Leriche syndrome Leukocyte-endothelial cell adhesion molecules (LECAM) Leukocytoclastic cutaneous vasculitis Limb apraxia Limb-girdle dystrophy Liver Liver abscesses Liver disease Liver failure Ludwig's angina Lumbar adhesive arachnoiditis, back pain from Lung cancer Lung disease Lymphoblastic leukemia Lymphoblastic lymphoma, precursor T cell/B cell Lymphoma(s) Lymphoma cutis Lysyl hydroxylase deficiency Labetalol [ch70 ¶85] [ch70 Table1] for aortic dissection [ch247 ¶20] dosage [ch70 Table1] for hypertension [ch246 ¶64] [ch246 Table4] malignant [ch246 ¶91] poisoning/overdosage [ch391 ¶80] for preeclampsia [ch7 ¶9] Laboratory tests [ch1 ¶22] [ch3 ¶10-¶22] false positive [ch3 ¶11] indications, accuracy, and usefulness of [ch3 ¶10-¶22] integration of clinical data and [ch3 ¶16-¶22] [ch3 Table4] for occupational/environmental hazards [ch5 ¶10] receiver operating characteristic curve [ch3 Figure1] [ch3 ¶14] terminology of [ch3 Table2] true positive [ch3 ¶11] Labyrinthine dysfunction acute bilateral [ch20 ¶64] acute unilateral [ch20 ¶62] recurrent unilateral [ch20 ¶65] and vertigo [ch20 ¶60] [ch372 ¶27] Labyrinthitis acute [ch20 ¶62] purulent [ch372 ¶27] serous [ch372 ¶27] Lacidipine, poisoning/overdosage [ch391 ¶85]

60. YO Pospishil, TM Antonovych were found focal glomerulonephritis with crescents (FGN) (5 cases), acute tubulointerstitialdiseases (ATID) (8 cases) and lipoid nephrosis with tubulo http://psk2.amu.edu.pl/CZASOPIS/PATP49-1/PJ49-5.HTM

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